Gaia Pedriali

Cotignola, Italy

Last Updated: 09/26/2026

Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 29 publications

Mitochondrial permeability transition pore desensitization by a novel dispiranic derivative prevents cardiac reperfusion injury.

Mitochondrial permeability transition pore desensitization by a novel dispiranic derivative prevents cardiac reperfusion injury.

Journal: Redox biology
Published: February 05, 2026

Mitochondria at the Crossroads of Cardiovascular Disease: Mechanistic Drivers and Emerging Therapeutic Strategies.

Mitochondria at the Crossroads of Cardiovascular Disease: Mechanistic Drivers and Emerging Therapeutic Strategies.

Journal: Cells
Published: December 30, 2025

Melatonin and mitochondrial protection in cardiac ischemia-reperfusion injury: mechanisms, evidence and translational perspectives.

Melatonin and mitochondrial protection in cardiac ischemia-reperfusion injury: mechanisms, evidence and translational perspectives.

Journal: Basic research in cardiology
Published: October 10, 2025

Design and synthesis of 1,4,8-triazaspiro[4.5]decan-2-one derivatives as novel mitochondrial permeability transition pore inhibitors.

Design and synthesis of 1,4,8-triazaspiro[4.5]decan-2-one derivatives as novel mitochondrial permeability transition pore inhibitors.

Journal: Journal of enzyme inhibition and medicinal chemistry
Published: May 21, 2025

Melatonin rescues cell respiration impaired by hypoxia/reoxygenation in aortic endothelial cells and affects the mitochondrial bioenergetics targeting the F1FO-ATPase.

Melatonin rescues cell respiration impaired by hypoxia/reoxygenation in aortic endothelial cells and affects the mitochondrial bioenergetics targeting the F1FO-ATPase.

Journal: Redox biology
Published: March 17, 2025

Calcium signaling from sarcoplasmic reticulum and mitochondria contact sites in acute myocardial infarction.

Calcium signaling from sarcoplasmic reticulum and mitochondria contact sites in acute myocardial infarction.

Journal: Journal of translational medicine
Published: February 29, 2024

ErbB2-NOTCH1 axis controls autophagy in cardiac cells.

ErbB2-NOTCH1 axis controls autophagy in cardiac cells.

Journal: BioFactors (Oxford, England)
Published: November 30, 2023

The Complex Relationship between Hypoxia Signaling, Mitochondrial Dysfunction and Inflammation in Calcific Aortic Valve Disease: Insights from the Molecular Mechanisms to Therapeutic Approaches.

The Complex Relationship between Hypoxia Signaling, Mitochondrial Dysfunction and Inflammation in Calcific Aortic Valve Disease: Insights from the Molecular Mechanisms to Therapeutic Approaches.

Journal: International journal of molecular sciences
Published: May 23, 2023

1,3,8-Triazaspiro[4.5]decane Derivatives Inhibit Permeability Transition Pores through a FO-ATP Synthase c Subunit Glu119-Independent Mechanism That Prevents Oligomycin A-Related Side Effects.

1,3,8-Triazaspiro[4.5]decane Derivatives Inhibit Permeability Transition Pores through a FO-ATP Synthase c Subunit Glu119-Independent Mechanism That Prevents Oligomycin A-Related Side Effects.

Journal: International journal of molecular sciences
Published: February 02, 2023

Mitochondrial dysfunction and skeletal muscle atrophy: Causes, mechanisms, and treatment strategies.

Mitochondrial dysfunction and skeletal muscle atrophy: Causes, mechanisms, and treatment strategies.

Journal: Mitochondrion
Published: January 27, 2023

Comprehensive Analysis of Mitochondrial Dynamics Alterations in Heart Diseases.

Comprehensive Analysis of Mitochondrial Dynamics Alterations in Heart Diseases.

Journal: International journal of molecular sciences
Published: January 04, 2023

Perspectives on mitochondrial relevance in cardiac ischemia/reperfusion injury.

Perspectives on mitochondrial relevance in cardiac ischemia/reperfusion injury.

Journal: Frontiers in cell and developmental biology
Published: October 27, 2022
Showing 1-12 of 29

Last Updated: 09/26/2026