Gemma Comes
Barcelona, CT, Spain
Last Updated: 09/26/2026
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Found 23 publications
Chronic central-targeted Interleukin-6 overexpression promotes hippocampal and cortical neuropathology in the Tg2576 mouse model of Alzheimer's disease.
Journal: Alzheimer's research & therapy
Published: January 13, 2026
Correction: Characterization of the role of the antioxidant proteins metallothioneins 1 and 2 in an animal model of Alzheimer's disease.
Journal: Cellular and molecular life sciences : CMLS
Published: October 16, 2025
Correction: Characterization of the role of the antioxidant proteins Metallothioneins 1 and 2 in an animal model of alzheimer's disease.
Journal: Cellular and molecular life sciences : CMLS
Published: October 15, 2025
Interleukin-6-elicited chronic neuroinflammation may decrease survival but is not sufficient to drive disease progression in a mouse model of Leigh syndrome.
Journal: Journal of inflammation (London, England)
Published: August 10, 2023
Microglial response promotes neurodegeneration in the Ndufs4 KO mouse model of Leigh syndrome.
Journal: Glia
Published: November 26, 2021
A new mouse model to study restoration of interleukin-6 (IL-6) expression in a Cre-dependent manner: microglial IL-6 regulation of experimental autoimmune encephalomyelitis.
Journal: Journal of neuroinflammation
Published: July 22, 2020
Interleukin-6 Derived from the Central Nervous System May Influence the Pathogenesis of Experimental Autoimmune Encephalomyelitis in a Cell-Dependent Manner.
Journal: Cells
Published: December 09, 2019
IL-6 trans-signaling in the brain influences the behavioral and physio-pathological phenotype of the Tg2576 and 3xTgAD mouse models of Alzheimer's disease.
Journal: Brain, behavior, and immunity
Published: February 07, 2019
Mouse metallothionein-1 and metallothionein-2 are not biologically interchangeable in an animal model of multiple sclerosis, EAE.
Journal: Metallomics : integrated biometal science
Published: December 14, 2018
Different Responses to a High-Fat Diet in IL-6 Conditional Knockout Mice Driven by Constitutive GFAP-Cre and Synapsin 1-Cre Expression.
Journal: Neuroendocrinology
Published: July 19, 2018
Last Updated: 09/26/2026