Georg Reiser
Leipziger Str. 44
Magdeburg, ST 39120, Germany
Magdeburg, ST 39120, Germany
Last Updated: 09/26/2026
Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 50 publications
How the brain fights fatty acids' toxicity.
Journal: Neurochemistry international
Published: January 08, 2021
Toll-like receptors control p38 and JNK MAPK signaling pathways in rat astrocytes differently, when cultured in normal or high glucose concentrations.
Journal: Neurochemistry international
Published: March 14, 2019
Astrocytes synthesize primary and cyclopentenone prostaglandins that are negative regulators of their proliferation.
Journal: Biochemical and biophysical research communications
Published: March 27, 2018
Regulation of the ARE-binding proteins, TTP (tristetraprolin) and HuR (human antigen R), in inflammatory response in astrocytes.
Journal: Neurochemistry international
Published: November 12, 2017
The small heat shock proteins, especially HspB4 and HspB5 are promising protectants in neurodegenerative diseases.
Journal: Neurochemistry international
Published: October 24, 2017
Brain energy metabolism spurns fatty acids as fuel due to their inherent mitotoxicity and potential capacity to unleash neurodegeneration.
Journal: Neurochemistry international
Published: February 19, 2017
Recent progress in translational research on neurovascular and neurodegenerative disorders.
Journal: Restorative neurology and neuroscience
Published: January 07, 2017
Neurons and astrocytes in an infantile neuroaxonal dystrophy (INAD) mouse model show characteristic alterations in glutamate-induced Ca2+ signaling.
Journal: Neurochemistry international
Published: December 18, 2016
Inhibition of β-oxidation is not a valid therapeutic tool for reducing oxidative stress in conditions of neurodegeneration.
Journal: Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism
Published: April 09, 2016
Mitochondria from a mouse model of the human infantile neuroaxonal dystrophy (INAD) with genetic defects in VIA iPLA2 have disturbed Ca(2+) regulation with reduction in Ca(2+) capacity.
Journal: Neurochemistry international
Published: March 11, 2016
Mitochondrial Ca(2+) Processing by a Unit of Mitochondrial Ca(2+) Uniporter and Na(+)/Ca(2+) Exchanger Supports the Neuronal Ca(2+) Influx via Activated Glutamate Receptors.
Journal: Neurochemical research
Published: October 15, 2015
Last Updated: 09/26/2026