Gigliola Reato
Turin, Italy
Last Updated: 09/26/2026
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Found 8 publications
MicroRNA 483-3p overexpression unleashes invasive growth of metastatic colorectal cancer via NDRG1 downregulation and ensuing activation of the ERBB3/AKT axis.
Journal: Molecular oncology
Published: November 04, 2022
Coexisting cancer stem cells with heterogeneous gene amplifications, transcriptional profiles, and malignancy are isolated from single glioblastomas.
Journal: Cell reports
Published: September 16, 2022
ERBB3 overexpression due to miR-205 inactivation confers sensitivity to FGF, metabolic activation, and liability to ERBB3 targeting in glioblastoma.
Journal: Cell reports
Published: September 21, 2020
A Molecularly Annotated Model of Patient-Derived Colon Cancer Stem-Like Cells to Assess Genetic and Nongenetic Mechanisms of Resistance to Anti-EGFR Therapy.
Journal: Clinical cancer research : an official journal of the American Association for Cancer Research
Published: July 27, 2017
MET inhibition overcomes radiation resistance of glioblastoma stem-like cells.
Journal: EMBO molecular medicine
Published: May 04, 2016
TNF-α promotes invasive growth through the MET signaling pathway.
Journal: Molecular oncology
Published: August 01, 2014
MET signaling in colon cancer stem-like cells blunts the therapeutic response to EGFR inhibitors.
Journal: Cancer research
Published: January 23, 2014
The MET oncogene is a functional marker of a glioblastoma stem cell subtype.
Journal: Cancer research
Published: June 29, 2012
Showing 1-8 of 8
Last Updated: 09/26/2026