Giulia Campostrini
Milan, Italy
Last Updated: 09/26/2026
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Found 19 publications
Interplay between calcium and sarcomeres directs cardiomyocyte maturation during regeneration.
Journal: Science (New York, N.Y.)
Published: May 18, 2023
Lack of the transcription factor Nfix causes tachycardia in mice sinus node and rats neonatal cardiomyocytes.
Journal: Acta physiologica (Oxford, England)
Published: February 05, 2023
RagD auto-activating mutations impair MiT/TFE activity in kidney tubulopathy and cardiomyopathy syndrome.
Journal: Nature communications
Published: June 02, 2022
Maturation of hiPSC-derived cardiomyocytes promotes adult alternative splicing of SCN5A and reveals changes in sodium current associated with cardiac arrhythmia.
Journal: Cardiovascular research
Published: October 19, 2021
Cardiac Tissues From Stem Cells: New Routes to Maturation and Cardiac Regeneration.
Journal: Circulation research
Published: March 18, 2021
A detailed characterization of the hyperpolarization-activated "funny" current (If) in human-induced pluripotent stem cell (iPSC)-derived cardiomyocytes with pacemaker activity.
Journal: Pflugers Archiv : European journal of physiology
Published: October 22, 2020
Generation, functional analysis and applications of isogenic three-dimensional self-aggregating cardiac microtissues from human pluripotent stem cells.
Journal: Nature protocols
Published: July 23, 2020
Targeting the Kv11.1 (hERG) channel with allosteric modulators. Synthesis and biological evaluation of three novel series of LUF7346 derivatives.
Journal: European journal of medicinal chemistry
Published: July 17, 2020
Human-iPSC-Derived Cardiac Stromal Cells Enhance Maturation in 3D Cardiac Microtissues and Reveal Non-cardiomyocyte Contributions to Heart Disease.
Journal: Cell stem cell
Published: February 03, 2020
Human iPSC modelling of a familial form of atrial fibrillation reveals a gain of function of If and ICaL in patient-derived cardiomyocytes.
Journal: Cardiovascular research
Published: November 15, 2018
A Loss-of-Function HCN4 Mutation Associated With Familial Benign Myoclonic Epilepsy in Infancy Causes Increased Neuronal Excitability.
Journal: Frontiers in molecular neuroscience
Published: March 05, 2018
Last Updated: 09/26/2026