Gloria Garoffolo
Via C. Parea, 4
Milan, 20138, Italy
Milan, 20138, Italy
Last Updated: 09/26/2026
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Found 18 publications
Blockade of YAP Mechanoactivation Prevents Neointima Formation and Adverse Remodeling in Arterialized Vein Grafts.
Journal: Journal of the American Heart Association
Published: March 21, 2025
When cell mechanics meets epitranscriptomics: reduction of m6A in Piezo2 RNA ameliorates cardiac fibrosis.
Journal: Cardiovascular research
Published: December 10, 2024
Reversion of aortic valve cells calcification by activation of Notch signalling via histone acetylation induction.
Journal: Signal transduction and targeted therapy
Published: August 14, 2024
Cells and Materials for Cardiac Repair and Regeneration.
Journal: Journal of clinical medicine
Published: April 11, 2023
Relevance of Spike/Estrogen Receptor-α interaction for endothelial-based coagulopathy induced by SARS-CoV-2.
Journal: Signal transduction and targeted therapy
Published: January 02, 2023
Reduction of Cardiac Fibrosis by Interference With YAP-Dependent Transactivation.
Journal: Circulation research
Published: June 30, 2022
Mechanosensor YAP cooperates with TGF-β1 signaling to promote myofibroblast activation and matrix stiffening in a 3D model of human cardiac fibrosis.
Journal: Acta biomaterialia
Published: April 15, 2022
Mechanical Strain Induces Transcriptomic Reprogramming of Saphenous Vein Progenitors.
Journal: Frontiers in cardiovascular medicine
Published: March 02, 2022
Vascular dysfunction and pathology: focus on mechanical forces.
Journal: Vascular biology (Bristol, England)
Published: May 24, 2021
From dissection of fibrotic pathways to assessment of drug interactions to reduce cardiac fibrosis and heart failure.
Journal: Current research in pharmacology and drug discovery
Published: March 02, 2021
A fluorogenic peptide-based smartprobe for the detection of neutrophil extracellular traps and inflammation.
Journal: Chemical communications (Cambridge, England)
Published: December 17, 2020
Human cardiosphere-derived stromal cells exposed to SARS-CoV-2 evolve into hyper-inflammatory/pro-fibrotic phenotype and produce infective viral particles depending on the levels of ACE2 receptor expression.
Journal: Cardiovascular research
Published: November 23, 2020
Last Updated: 09/26/2026