Guillaume Collin

Lyon, 69008, France

Last Updated: 09/26/2026

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Found 8 publications

Intestinal Epithelial Cells Adapt to Chronic Inflammation through Partial Genetic Reprogramming.

Intestinal Epithelial Cells Adapt to Chronic Inflammation through Partial Genetic Reprogramming.

Journal: Cancers
Published: January 12, 2023

Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A.

Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A.

Journal: Cancer research
Published: December 19, 2019

Calcium channel ITPR2 and mitochondria-ER contacts promote cellular senescence and aging.

Calcium channel ITPR2 and mitochondria-ER contacts promote cellular senescence and aging.

Journal: Nature communications
Published: August 26, 2019

The antioxidant N-acetylcysteine protects from lung emphysema but induces lung adenocarcinoma in mice.

The antioxidant N-acetylcysteine protects from lung emphysema but induces lung adenocarcinoma in mice.

Journal: JCI insight
Published: January 24, 2019

Transcriptional repression of DNA repair genes is a hallmark and a cause of cellular senescence.

Transcriptional repression of DNA repair genes is a hallmark and a cause of cellular senescence.

Journal: Cell death & disease
Published: December 08, 2017

The Heterodimeric TWIST1-E12 Complex Drives the Oncogenic Potential of TWIST1 in Human Mammary Epithelial Cells.

The Heterodimeric TWIST1-E12 Complex Drives the Oncogenic Potential of TWIST1 in Human Mammary Epithelial Cells.

Journal: Neoplasia (New York, N.Y.)
Published: December 22, 2015

Senescence versus apoptosis in chemotherapy.

Senescence versus apoptosis in chemotherapy.

Journal: Oncotarget
Published: March 04, 2015

EMT or EMT-Promoting Transcription Factors, Where to Focus the Light?

EMT or EMT-Promoting Transcription Factors, Where to Focus the Light?

Journal: Frontiers in oncology
Published: September 30, 2014
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Last Updated: 09/26/2026