Guoyu Zhao
Beijing, China
Last Updated: 09/26/2026
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Found 17 publications
Prognostic Value and Potential Mechanism of CD8+ T Subsets Phenotype in ICU Patients with Sepsis.
Journal: Shock (Augusta, Ga.)
Published: January 30, 2026
Restricted TCR Diversity and Expansion of CD177+CD8+ and TLR4+CD4+T Cell Subsets in Septic Shock-Associated Immunosuppression: A Single-Cell Multi-Omics Study.
Journal: MedComm
Published: January 23, 2026
Frailty Index-laboratory and lymphocyte subset patterns in predicting 28-day mortality among elderly sepsis patients: a multicenter observational cohort study.
Journal: Frontiers in immunology
Published: May 07, 2025
Dynamic alterations of lymphocyte subsets following sepsis in octogenarian and elder patients.
Journal: Journal of intensive medicine
Published: January 07, 2025
Corrigendum to "mTOR aggravated CD4+ T cell pyroptosis by regulating the PPARγ-Nrf2 pathway in sepsis" [Int. Immunopharmacol. 140 (2024) 112822].
Journal: International immunopharmacology
Published: October 15, 2024
mTOR pathway mediates the endoplasmic reticulum stress -apoptosis of CD4+ T cell through inhibiting autophagy flux in sepsis.
Journal: Inflammation research : official journal of the European Histamine Research Society ... [et al.]
Published: September 28, 2024
Predicting intra-abdominal candidiasis in elderly septic patients using machine learning based on lymphocyte subtyping: a prospective cohort study.
Journal: Frontiers in pharmacology
Published: August 26, 2024
Establishment and Validation of a Machine-Learning Prediction Nomogram Based on Lymphocyte Subtyping for Intra-Abdominal Candidiasis in Septic Patients.
Journal: Clinical and translational science
Published: August 16, 2024
Dynamic changes in peripheral blood lymphocyte trajectory predict the clinical outcomes of sepsis.
Journal: Frontiers in immunology
Published: May 11, 2024
mTOR aggravated CD4+ T cell pyroptosis by regulating the PPARγ-Nrf2 pathway in sepsis.
Journal: International immunopharmacology
Published: May 05, 2024
mTOR Deletion Alleviates CD4+ T-Cell Dysfunction in Sepsis through Reducing CTLA4 Accumulation Mediated by Rescuing Autophagy.
Journal: Mediators of inflammation
Published: April 20, 2024
Last Updated: 09/26/2026