Hahyun Park

Seoul, Republic of Korea

Last Updated: 09/26/2026

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Found 32 publications

CRISPR/Cas9-mediated SLC2A1 gene knockout changes in energy metabolism and cellular behavior in human trophoblasts.

CRISPR/Cas9-mediated SLC2A1 gene knockout changes in energy metabolism and cellular behavior in human trophoblasts.

Journal: Reproduction (Cambridge, England)

Published: September 01, 2025

Disruption of mitochondrial homeostasis and apoptosis by oryzalin exposure in zebrafish embryos.

Disruption of mitochondrial homeostasis and apoptosis by oryzalin exposure in zebrafish embryos.

Journal: Comparative biochemistry and physiology. Toxicology & pharmacology : CBP

Published: March 18, 2025

Exposure to acifluorfen induces developmental toxicity in the early life stage of zebrafish.

Exposure to acifluorfen induces developmental toxicity in the early life stage of zebrafish.

Journal: Comparative biochemistry and physiology. Toxicology & pharmacology : CBP

Published: December 27, 2023

Bifenox induces hepatotoxicity and vascular toxicity in zebrafish embryos via ROS production and alterations in signaling pathways.

Bifenox induces hepatotoxicity and vascular toxicity in zebrafish embryos via ROS production and alterations in signaling pathways.

Journal: Comparative biochemistry and physiology. Toxicology & pharmacology : CBP

Published: October 03, 2023

Bifenox compromises porcine trophectoderm and luminal epithelial cells in early pregnancy by arresting cell cycle progression and impairing mitochondrial and calcium homeostasis.

Bifenox compromises porcine trophectoderm and luminal epithelial cells in early pregnancy by arresting cell cycle progression and impairing mitochondrial and calcium homeostasis.

Journal: Pesticide biochemistry and physiology

Published: July 19, 2023

Dimethenamid promotes oxidative stress and apoptosis leading to cardiovascular, hepatic, and pancreatic toxicities in zebrafish embryo.

Dimethenamid promotes oxidative stress and apoptosis leading to cardiovascular, hepatic, and pancreatic toxicities in zebrafish embryo.

Journal: Comparative biochemistry and physiology. Toxicology & pharmacology : CBP

Published: July 11, 2023

Developmental toxicity of flufenacet including vascular, liver, and pancreas defects is mediated by apoptosis and alters the Mapk and PI3K/Akt signal transduction in zebrafish.

Developmental toxicity of flufenacet including vascular, liver, and pancreas defects is mediated by apoptosis and alters the Mapk and PI3K/Akt signal transduction in zebrafish.

Journal: Comparative biochemistry and physiology. Toxicology & pharmacology : CBP

Published: April 05, 2023

Fluchloralin induces developmental toxicity in heart, liver, and nervous system during early zebrafish embryogenesis.

Fluchloralin induces developmental toxicity in heart, liver, and nervous system during early zebrafish embryogenesis.

Journal: Comparative biochemistry and physiology. Toxicology & pharmacology : CBP

Published: March 21, 2023

Developmental defects induced by thiabendazole are mediated via apoptosis, oxidative stress and alteration in PI3K/Akt and MAPK pathways in zebrafish.

Developmental defects induced by thiabendazole are mediated via apoptosis, oxidative stress and alteration in PI3K/Akt and MAPK pathways in zebrafish.

Journal: Environment international

Published: January 30, 2023

Embryonic exposure to chloroxylenol induces developmental defects and cardiovascular toxicity via oxidative stress, inflammation, and apoptosis in zebrafish.

Embryonic exposure to chloroxylenol induces developmental defects and cardiovascular toxicity via oxidative stress, inflammation, and apoptosis in zebrafish.

Journal: Comparative biochemistry and physiology. Toxicology & pharmacology : CBP

Published: December 31, 2022

Establishment of Immortalized Human Endometriotic Stromal Cell Line from Ectopic Lesion of a Patient with Endometriosis.

Establishment of Immortalized Human Endometriotic Stromal Cell Line from Ectopic Lesion of a Patient with Endometriosis.

Journal: Reproductive sciences (Thousand Oaks, Calif.)

Published: November 30, 2022

Targeting Thymidylate Synthase and tRNA-Derived Non-Coding RNAs Improves Therapeutic Sensitivity in Colorectal Cancer.

Targeting Thymidylate Synthase and tRNA-Derived Non-Coding RNAs Improves Therapeutic Sensitivity in Colorectal Cancer.

Journal: Antioxidants (Basel, Switzerland)

Published: September 21, 2022

Showing 1-12 of 32

Last Updated: 09/26/2026