Hailin Zhang

Key Laboratory Of Animal Models And Human Disease Mechanisms Of The Chinese Academy Of Sciences And Yunnan Province
Kunming, 650223, China

Last Updated: 09/26/2026

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Found 14 publications

HECTD3 is overexpressed in breast cancer and associated with a good prognosis.

HECTD3 is overexpressed in breast cancer and associated with a good prognosis.

Journal: Acta biochimica et biophysica Sinica
Published: June 14, 2025

Correction: BRD4-specific PROTAC inhibits basal-like breast cancer partially through downregulating KLF5 expression.

Correction: BRD4-specific PROTAC inhibits basal-like breast cancer partially through downregulating KLF5 expression.

Journal: Oncogene
Published: February 03, 2025

Adaptive radiation and social evolution of the ants.

Adaptive radiation and social evolution of the ants.

Journal: Cell
Published: December 04, 2024

BRD4-specific PROTAC inhibits basal-like breast cancer partially through downregulating KLF5 expression.

BRD4-specific PROTAC inhibits basal-like breast cancer partially through downregulating KLF5 expression.

Journal: Oncogene
Published: December 22, 2023

Targeting HECTD3-IKKα axis inhibits inflammation-related metastasis.

Targeting HECTD3-IKKα axis inhibits inflammation-related metastasis.

Journal: Signal transduction and targeted therapy
Published: September 23, 2021

Characterization of tree shrew telomeres and telomerase.

Characterization of tree shrew telomeres and telomerase.

Journal: Journal of genetics and genomics = Yi chuan xue bao
Published: March 13, 2021

USP3 promotes breast cancer cell proliferation by deubiquitinating KLF5.

USP3 promotes breast cancer cell proliferation by deubiquitinating KLF5.

Journal: The Journal of biological chemistry
Published: April 27, 2019

Glucocorticoid Receptor Signaling Activates TEAD4 to Promote Breast Cancer Progression.

Glucocorticoid Receptor Signaling Activates TEAD4 to Promote Breast Cancer Progression.

Journal: Cancer research
Published: January 02, 2019

Mithramycin A suppresses basal triple-negative breast cancer cell survival partially via down-regulating Krüppel-like factor 5 transcription by Sp1.

Mithramycin A suppresses basal triple-negative breast cancer cell survival partially via down-regulating Krüppel-like factor 5 transcription by Sp1.

Journal: Scientific reports
Published: November 02, 2017

Hypoxia induces miR-153 through the IRE1α-XBP1 pathway to fine tune the HIF1α/VEGFA axis in breast cancer angiogenesis.

Hypoxia induces miR-153 through the IRE1α-XBP1 pathway to fine tune the HIF1α/VEGFA axis in breast cancer angiogenesis.

Journal: Oncogene
Published: June 13, 2017

Metformin suppresses triple-negative breast cancer stem cells by targeting KLF5 for degradation.

Metformin suppresses triple-negative breast cancer stem cells by targeting KLF5 for degradation.

Journal: Cell discovery
Published: October 27, 2016

The interplay between TEAD4 and KLF5 promotes breast cancer partially through inhibiting the transcription of p27Kip1.

The interplay between TEAD4 and KLF5 promotes breast cancer partially through inhibiting the transcription of p27Kip1.

Journal: Oncotarget
Published: January 10, 2015
Showing 1-12 of 14

Last Updated: 09/26/2026