Haitao Li
State Key Laboratory Of Molecular Oncology, MOE Key Laboratory Of Protein Sciences
Beijing, 100084, China
Beijing, 100084, China
Last Updated: 09/26/2026
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Found 12 publications
YEATS4 reads histone crotonylation to promote fatty acid metabolism and cancer cell stemness.
Journal: Cell reports
Published: March 04, 2025
KMT2D-mediated H3K4me1 recruits YBX1 to facilitate triple-negative breast cancer progression through epigenetic activation of c-Myc.
Journal: Clinical and translational medicine
Published: March 24, 2024
Diffuse midline glioma treated with epigenetic agent-based immunotherapy.
Journal: Signal transduction and targeted therapy
Published: August 12, 2022
Histone Modifications and Chondrocyte Fate: Regulation and Therapeutic Implications.
Journal: Frontiers in cell and developmental biology
Published: November 06, 2020
Cancer-driving H3G34V/R/D mutations block H3K36 methylation and H3K36me3-MutSα interaction.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: September 06, 2018
PTEN Suppresses Glycolysis by Dephosphorylating and Inhibiting Autophosphorylated PGK1.
Journal: Molecular cell
Published: September 06, 2018
The histone mark H3K36me2 recruits DNMT3A and shapes the intergenic DNA methylation landscape.
Journal: Nature
Published: August 30, 2018
Histone H1 defect in escort cells triggers germline tumor in Drosophila ovary.
Journal: Developmental biology
Published: September 16, 2016
ZMYND11: an H3.3-specific reader of H3K36me3.
Journal: Cell cycle (Georgetown, Tex.)
Published: June 26, 2014
ZMYND11 links histone H3.3K36me3 to transcription elongation and tumour suppression.
Journal: Nature
Published: January 27, 2013
Showing 1-12 of 12
Last Updated: 09/26/2026