Haixin Ju
Shanghai, 201102, China
Last Updated: 09/26/2026
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Found 9 publications
Vitamin D Deficiency Activates Gdnf-Ret-pErk1/2 Signal and Induces Kidney Malformations in Mice.
Journal: International journal of molecular sciences
Published: February 01, 2026
Excessive maternal methionine intake increases the incidence of congenital anomalies of the kidney and urinary tract in offspring mice.
Journal: Nutrition research (New York, N.Y.)
Published: January 21, 2026
Maternal Exposure to 2,4-Di-tert-butylphenol During Pregnancy in a Mouse Model Leads to Abnormal Development of the Urinary System in Offspring.
Journal: Toxics
Published: October 21, 2025
Vitamin A Deficiency Disturbs Ret Expression and Induces Urinary Tract Developmental Abnormalities in Mice.
Journal: American journal of nephrology
Published: July 22, 2024
Gestational diabetes mellitus induces congenital anomalies of the kidney and urinary tract in mice by altering RET/MAPK/ERK pathway.
Journal: Biochemical and biophysical research communications
Published: April 05, 2024
GEN1 as a risk factor for human congenital anomalies of the kidney and urinary tract.
Journal: Human genomics
Published: October 15, 2023
Overexpression of long noncoding RNA 4933425B07Rik leads to renal hypoplasia by inactivating Wnt/β-catenin signaling pathway.
Journal: Frontiers in cell and developmental biology
Published: August 10, 2023
Factors associated with postoperative muscle reconnection in children's congenital muscular torticollis.
Journal: Translational pediatrics
Published: March 10, 2023
Inhibition of MAPK/ERK pathway activation rescues congenital anomalies of the kidney and urinary tract (CAKUT) in Robo2PB/+ Gen1PB/+ mice.
Journal: Biochemical and biophysical research communications
Published: February 08, 2023
Showing 1-9 of 9
Last Updated: 09/26/2026