Hamid Nikbakht
Montreal, QC H3A1B1, Canada
Last Updated: 09/26/2026
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Found 18 publications
H3K27M in Gliomas Causes a One-Step Decrease in H3K27 Methylation and Reduced Spreading within the Constraints of H3K36 Methylation.
Journal: Cell reports
Published: April 29, 2020
Author Correction: Germline HAVCR2 mutations altering TIM-3 characterize subcutaneous panniculitis-like T cell lymphomas with hemophagocytic lymphohistiocytic syndrome.
Journal: Nature genetics
Published: November 16, 2018
H3K27M induces defective chromatin spread of PRC2-mediated repressive H3K27me2/me3 and is essential for glioma tumorigenesis.
Journal: Nature communications
Published: November 07, 2018
The histone mark H3K36me2 recruits DNMT3A and shapes the intergenic DNA methylation landscape.
Journal: Nature
Published: August 30, 2018
Methylome analysis and whole-exome sequencing reveal that brain tumors associated with encephalocraniocutaneous lipomatosis are midline pilocytic astrocytomas.
Journal: Acta neuropathologica
Published: April 25, 2018
TRPV4 and KRAS and FGFR1 gain-of-function mutations drive giant cell lesions of the jaw.
Journal: Nature communications
Published: March 13, 2018
Germline HAVCR2 mutations altering TIM-3 characterize subcutaneous panniculitis-like T cell lymphomas with hemophagocytic lymphohistiocytic syndrome.
Journal: Nature genetics
Published: March 05, 2018
Latency and interval therapy affect the evolution in metastatic colorectal cancer.
Journal: Scientific reports
Published: October 19, 2017
Characterizing temporal genomic heterogeneity in pediatric high-grade gliomas.
Journal: Acta neuropathologica communications
Published: October 04, 2017
Longitudinal mutational analysis of a cerebellar pilocytic astrocytoma recurring as a ganglioglioma.
Longitudinal mutational analysis of a cerebellar pilocytic astrocytoma recurring as a ganglioglioma.
Journal: Pediatric blood & cancer
Published: April 19, 2016
Last Updated: 09/26/2026