Hanna Erdmann

Hamburg, HH, Germany

Last Updated: 09/26/2026

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Found 13 publications

Correction to: Interleukin‑23 instructs protective multifunctional CD4 T cell responses after immunization with the Mycobacterium tuberculosis subunit vaccine H1 DDA/TDB independently of interleukin‑17A.

Correction to: Interleukin‑23 instructs protective multifunctional CD4 T cell responses after immunization with the Mycobacterium tuberculosis subunit vaccine H1 DDA/TDB independently of interleukin‑17A.

Journal: Journal of molecular medicine (Berlin, Germany)

Published: September 27, 2021

Peroxisome proliferator-activated receptor-γ signalling protects hair follicle stem cells from chemotherapy-induced apoptosis and epithelial-mesenchymal transition.

Peroxisome proliferator-activated receptor-γ signalling protects hair follicle stem cells from chemotherapy-induced apoptosis and epithelial-mesenchymal transition.

Journal: The British journal of dermatology

Published: August 28, 2021

Interleukin-23 instructs protective multifunctional CD4 T cell responses after immunization with the Mycobacterium tuberculosis subunit vaccine H1 DDA/TDB independently of interleukin-17A.

Interleukin-23 instructs protective multifunctional CD4 T cell responses after immunization with the Mycobacterium tuberculosis subunit vaccine H1 DDA/TDB independently of interleukin-17A.

Journal: Journal of molecular medicine (Berlin, Germany)

Published: December 09, 2020

A novel nondrug SFRP1 antagonist inhibits catagen development in human hair follicles ex vivo.

A novel nondrug SFRP1 antagonist inhibits catagen development in human hair follicles ex vivo.

Journal: The British journal of dermatology

Published: June 10, 2020

Preclinical evidence that the PPARγ modulator, N-Acetyl-GED-0507-34-Levo, may protect human hair follicle epithelial stem cells against lichen planopilaris-associated damage.

Preclinical evidence that the PPARγ modulator, N-Acetyl-GED-0507-34-Levo, may protect human hair follicle epithelial stem cells against lichen planopilaris-associated damage.

Journal: Journal of the European Academy of Dermatology and Venereology : JEADV

Published: November 28, 2019

De Novo Fatty Acid Synthesis During Mycobacterial Infection Is a Prerequisite for the Function of Highly Proliferative T Cells, But Not for Dendritic Cells or Macrophages.

De Novo Fatty Acid Synthesis During Mycobacterial Infection Is a Prerequisite for the Function of Highly Proliferative T Cells, But Not for Dendritic Cells or Macrophages.

Journal: Frontiers in immunology

Published: October 25, 2017

The increased protection and pathology in Mycobacterium tuberculosis-infected IL-27R-alpha-deficient mice is supported by IL-17A and is associated with the IL-17A-induced expansion of multifunctional T cells.

The increased protection and pathology in Mycobacterium tuberculosis-infected IL-27R-alpha-deficient mice is supported by IL-17A and is associated with the IL-17A-induced expansion of multifunctional T cells.

Journal: Mucosal immunology

Published: August 09, 2017

Suppressor of Cytokine Signaling 3 in Macrophages Prevents Exacerbated Interleukin-6-Dependent Arginase-1 Activity and Early Permissiveness to Experimental Tuberculosis.

Suppressor of Cytokine Signaling 3 in Macrophages Prevents Exacerbated Interleukin-6-Dependent Arginase-1 Activity and Early Permissiveness to Experimental Tuberculosis.

Journal: Frontiers in immunology

Published: August 08, 2017

During acute experimental infection with the reticulotropic Trypanosoma cruzi strain Tulahuen IL-22 is induced IL-23-dependently but is dispensable for protection.

During acute experimental infection with the reticulotropic Trypanosoma cruzi strain Tulahuen IL-22 is induced IL-23-dependently but is dispensable for protection.

Journal: Scientific reports

Published: April 11, 2016

Epstein-Barr virus-induced gene 3 suppresses T helper type 1, type 17 and type 2 immune responses after Trypanosoma cruzi infection and inhibits parasite replication by interfering with alternative macrophage activation.

Epstein-Barr virus-induced gene 3 suppresses T helper type 1, type 17 and type 2 immune responses after Trypanosoma cruzi infection and inhibits parasite replication by interfering with alternative macrophage activation.

Journal: Immunology

Published: July 23, 2015

9- and 11-Substituted 4-azapaullones are potent and selective inhibitors of African trypanosoma.

9- and 11-Substituted 4-azapaullones are potent and selective inhibitors of African trypanosoma.

Journal: European journal of medicinal chemistry

Published: March 31, 2014

The IL-13/IL-4Rα axis is involved in tuberculosis-associated pathology.

The IL-13/IL-4Rα axis is involved in tuberculosis-associated pathology.

Journal: The Journal of pathology

Published: February 13, 2014

Showing 1-12 of 13

Last Updated: 09/26/2026