Hanwei Jiang
443 Huangshan Road
Hefei, 230027, China
Hefei, 230027, China
Last Updated: 09/26/2026
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Found 27 publications
Synaptonemal complex: The structural basis for meiosis I.
Journal: Current topics in developmental biology
Published: May 07, 2026
Loss-of-Function Variants in CCDC189 Cause Human Oligoasthenoteratozoospermia by Disrupting Sperm Flagellar and Acrosomal Architecture.
Journal: Andrology
Published: February 10, 2026
Novel variants in YTHDC2 cause non-obstructive azoospermia by disrupting the mitotic-to-meiotic transition in humans and mice.
Journal: Human reproduction (Oxford, England)
Published: January 13, 2026
S100PBP interacts with nucleoporin TPR and facilitates XY crossover formation in mice.
Journal: EMBO reports
Published: June 21, 2024
A novel recombination protein C12ORF40/REDIC1 is required for meiotic crossover formation.
Journal: Cell discovery
Published: January 31, 2023
Loss-of-function variants in KCTD19 cause non-obstructive azoospermia in humans.
Journal: iScience
Published: December 20, 2022
M1AP interacts with the mammalian ZZS complex and promotes male meiotic recombination.
Journal: EMBO reports
Published: July 12, 2022
Homozygous mutations in C14orf39/SIX6OS1 cause non-obstructive azoospermia and premature ovarian insufficiency in humans.
Journal: American journal of human genetics
Published: July 08, 2022
Identification of pathogenic mutations from nonobstructive azoospermia patients†.
Journal: Biology of reproduction
Published: January 15, 2022
RAD51AP2 is required for efficient meiotic recombination between X and Y chromosomes.
Journal: Science advances
Published: January 12, 2022
Biallelic HFM1 variants cause non-obstructive azoospermia with meiotic arrest in humans by impairing crossover formation to varying degrees.
Journal: Human reproduction (Oxford, England)
Published: October 12, 2021
Last Updated: 09/26/2026