Hao Gao

Key Laboratory Of Biotechnology And Bioengineering Of State Ethnic Affairs Commission
Lanzhou, 730030, China

Last Updated: 09/26/2026

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Found 16 publications

TRIM13 Positively Regulates the NF-κB Signaling Pathway Induced by Encephalomyocarditis Virus.

TRIM13 Positively Regulates the NF-κB Signaling Pathway Induced by Encephalomyocarditis Virus.

Journal: Viruses

Published: March 06, 2026

Monomeric C-reactive Protein Exacerbates Neuronal Injury and Enhances Microglial Activation after Global Cerebral Ischemia in Mice.

Monomeric C-reactive Protein Exacerbates Neuronal Injury and Enhances Microglial Activation after Global Cerebral Ischemia in Mice.

Journal: Molecular neurobiology

Published: July 25, 2025

Diselenides as novel effective fluorescence quenchers to construct a two-photon fluorescent probe for thiols in a mouse stroke model.

Diselenides as novel effective fluorescence quenchers to construct a two-photon fluorescent probe for thiols in a mouse stroke model.

Journal: Chemical communications (Cambridge, England)

Published: January 08, 2025

Transcriptomic Analysis Divulges Differential Expressions of Microglial Genes After Microglial Repopulation in Mice.

Transcriptomic Analysis Divulges Differential Expressions of Microglial Genes After Microglial Repopulation in Mice.

Journal: International journal of molecular sciences

Published: December 12, 2024

Cholecystitis and cholangiocarcinoma: a two-sample Mendelian randomization study.

Cholecystitis and cholangiocarcinoma: a two-sample Mendelian randomization study.

Journal: BMC gastroenterology

Published: May 24, 2024

Intestinal Akkermansia muciniphila is Beneficial to Functional Recovery Following Ischemic Stroke.

Intestinal Akkermansia muciniphila is Beneficial to Functional Recovery Following Ischemic Stroke.

Journal: Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology

Published: February 28, 2024

Rapamycin Alleviates Neuronal Injury and Modulates Microglial Activation After Cerebral Ischemia.

Rapamycin Alleviates Neuronal Injury and Modulates Microglial Activation After Cerebral Ischemia.

Journal: Molecular neurobiology

Published: October 03, 2022

Depletion of Arg1-Positive Microglia/Macrophages Exacerbates Cerebral Ischemic Damage by Facilitating the Inflammatory Response.

Depletion of Arg1-Positive Microglia/Macrophages Exacerbates Cerebral Ischemic Damage by Facilitating the Inflammatory Response.

Journal: International journal of molecular sciences

Published: September 20, 2022

Baylis-Hillman Adducts as a Versatile Module for Constructing Fluorogenic Release System.

Baylis-Hillman Adducts as a Versatile Module for Constructing Fluorogenic Release System.

Journal: Journal of medicinal chemistry

Published: April 15, 2022

Differential Regulation of Microglial Activation in Response to Different Degree of Ischemia.

Differential Regulation of Microglial Activation in Response to Different Degree of Ischemia.

Journal: Frontiers in immunology

Published: October 10, 2021

Developmental exposure to environmental levels of cadmium induces neurotoxicity and activates microglia in zebrafish larvae: From the perspectives of neurobehavior and neuroimaging.

Developmental exposure to environmental levels of cadmium induces neurotoxicity and activates microglia in zebrafish larvae: From the perspectives of neurobehavior and neuroimaging.

Journal: Chemosphere

Published: August 25, 2021

Depletion of iNOS-positive inflammatory cells decelerates neuronal degeneration and alleviates cerebral ischemic damage by suppressing the inflammatory response.

Depletion of iNOS-positive inflammatory cells decelerates neuronal degeneration and alleviates cerebral ischemic damage by suppressing the inflammatory response.

Journal: Free radical biology & medicine

Published: June 14, 2021

Showing 1-12 of 16

Last Updated: 09/26/2026