Hitomi Tanaka

Sekimachi, 5113892, Japan

Last Updated: 09/26/2026

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Found 11 publications

Mechanistic Insights into Tyrosinase-Catalyzed Metabolism of Hydroquinone: Implications for the Etiology of Exogenous Ochronosis and Cytotoxicity to Melanocytes.

Mechanistic Insights into Tyrosinase-Catalyzed Metabolism of Hydroquinone: Implications for the Etiology of Exogenous Ochronosis and Cytotoxicity to Melanocytes.

Journal: International journal of molecular sciences

Published: October 06, 2025

Exogenous ochronosis by hydroquinone is not caused by inhibition of homogentisate dioxygenase but potentially by tyrosinase-catalysed metabolism of hydroquinone.

Exogenous ochronosis by hydroquinone is not caused by inhibition of homogentisate dioxygenase but potentially by tyrosinase-catalysed metabolism of hydroquinone.

Journal: The British journal of dermatology

Published: May 16, 2025

Pterostilbene, a Dimethyl Derivative of Resveratrol, Exerts Cytotoxic Effects on Melanin-Producing Cells through Metabolic Activation by Tyrosinase.

Pterostilbene, a Dimethyl Derivative of Resveratrol, Exerts Cytotoxic Effects on Melanin-Producing Cells through Metabolic Activation by Tyrosinase.

Journal: International journal of molecular sciences

Published: August 22, 2024

Sperm Size Decreases With Increasing Pheomelanin Pigmentation but Not With the Amount of Glutathione in the Barn Swallow.

Sperm Size Decreases With Increasing Pheomelanin Pigmentation but Not With the Amount of Glutathione in the Barn Swallow.

Journal: Zoological science

Published: December 15, 2023

Corrigendum to "DOPA pheomelanin is increased in nigral neuromelanin of Parkinson's disease" [Prog. Neurobiol. 223 (2023) 102414].

Corrigendum to "DOPA pheomelanin is increased in nigral neuromelanin of Parkinson's disease" [Prog. Neurobiol. 223 (2023) 102414].

Journal: Progress in neurobiology

Published: May 08, 2023

DOPA pheomelanin is increased in nigral neuromelanin of Parkinson's disease.

DOPA pheomelanin is increased in nigral neuromelanin of Parkinson's disease.

Journal: Progress in neurobiology

Published: May 20, 2022

Oxidative Transformations of 3,4-Dihydroxyphenylacetaldehyde Generate Potential Reactive Intermediates as Causative Agents for Its Neurotoxicity.

Oxidative Transformations of 3,4-Dihydroxyphenylacetaldehyde Generate Potential Reactive Intermediates as Causative Agents for Its Neurotoxicity.

Journal: International journal of molecular sciences

Published: September 30, 2021

The Oxidation of Equol by Tyrosinase Produces a Unique Di-ortho-Quinone: Possible Implications for Melanocyte Toxicity.

The Oxidation of Equol by Tyrosinase Produces a Unique Di-ortho-Quinone: Possible Implications for Melanocyte Toxicity.

Journal: International journal of molecular sciences

Published: July 22, 2021

The Oxidative Pathway to Dopamine-Protein Conjugates and Their Pro-Oxidant Activities: Implications for the Neurodegeneration of Parkinson's Disease.

The Oxidative Pathway to Dopamine-Protein Conjugates and Their Pro-Oxidant Activities: Implications for the Neurodegeneration of Parkinson's Disease.

Journal: International journal of molecular sciences

Published: April 16, 2019

The Pro-Oxidant Activity of Pheomelanin is Significantly Enhanced by UVA Irradiation: Benzothiazole Moieties Are More Reactive than Benzothiazine Moieties.

The Pro-Oxidant Activity of Pheomelanin is Significantly Enhanced by UVA Irradiation: Benzothiazole Moieties Are More Reactive than Benzothiazine Moieties.

Journal: International journal of molecular sciences

Published: September 03, 2018

Reduction of the nitro group to amine by hydroiodic acid to synthesize o-aminophenol derivatives as putative degradative markers of neuromelanin.

Reduction of the nitro group to amine by hydroiodic acid to synthesize o-aminophenol derivatives as putative degradative markers of neuromelanin.

Journal: Molecules (Basel, Switzerland)

Published: May 04, 2014

Showing 1-11 of 11

Last Updated: 09/26/2026