Hitoshi Murata

Okayama, Japan

Last Updated: 09/26/2026

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Found 46 publications

New mechanistic insight into SARM1 activation: TRIM32-mediated ubiquitination is a key lever that actuates SARM1's catalytic action, leading to axon degeneration and cell death.

New mechanistic insight into SARM1 activation: TRIM32-mediated ubiquitination is a key lever that actuates SARM1's catalytic action, leading to axon degeneration and cell death.

Journal: Frontiers in neural circuits

Published: March 30, 2026

Carnosol, a Rosemary Ingredient Discovered in a Screen for Inhibitors of SARM1-NAD+ Cleavage Activity, Ameliorates Symptoms of Peripheral Neuropathy.

Carnosol, a Rosemary Ingredient Discovered in a Screen for Inhibitors of SARM1-NAD+ Cleavage Activity, Ameliorates Symptoms of Peripheral Neuropathy.

Journal: Antioxidants (Basel, Switzerland)

Published: May 22, 2025

Enhanced design of pCMViR-TSC plasmid vector for sustainably high cargo gene expression in mammalian cells.

Enhanced design of pCMViR-TSC plasmid vector for sustainably high cargo gene expression in mammalian cells.

Journal: In vitro cellular & developmental biology. Animal

Published: October 06, 2024

Correction: S100A11 is involved in the progression of colorectal cancer through the desmosome-catenin-TCF signaling pathway.

Correction: S100A11 is involved in the progression of colorectal cancer through the desmosome-catenin-TCF signaling pathway.

Journal: In vitro cellular & developmental biology. Animal

Published: June 27, 2024

An S100A8/A9 Neutralizing Antibody Potently Ameliorates Contact Hypersensitivity and Atopic Dermatitis Symptoms.

An S100A8/A9 Neutralizing Antibody Potently Ameliorates Contact Hypersensitivity and Atopic Dermatitis Symptoms.

Journal: The Journal of investigative dermatology

Published: April 03, 2024

S100A11 is involved in the progression of colorectal cancer through the desmosome-catenin-TCF signaling pathway.

S100A11 is involved in the progression of colorectal cancer through the desmosome-catenin-TCF signaling pathway.

Journal: In vitro cellular & developmental biology. Animal

Published: March 06, 2024

Dissection of the signal transduction machinery responsible for the lysyl oxidase-like 4-mediated increase in invasive motility in triple-negative breast cancer cells: mechanistic insight into the integrin-β1-NF-κB-MMP9 axis.

Dissection of the signal transduction machinery responsible for the lysyl oxidase-like 4-mediated increase in invasive motility in triple-negative breast cancer cells: mechanistic insight into the integrin-β1-NF-κB-MMP9 axis.

Journal: Frontiers in oncology

Published: February 07, 2024

Lysyl oxidase-like 4 promotes the invasiveness of triple-negative breast cancer cells by orchestrating the invasive machinery formed by annexin A2 and S100A11 on the cell surface.

Lysyl oxidase-like 4 promotes the invasiveness of triple-negative breast cancer cells by orchestrating the invasive machinery formed by annexin A2 and S100A11 on the cell surface.

Journal: Frontiers in oncology

Published: January 16, 2024

Phosphorylated SARM1 is involved in the pathological process of rotenone-induced neurodegeneration.

Phosphorylated SARM1 is involved in the pathological process of rotenone-induced neurodegeneration.

Journal: Journal of biochemistry

Published: May 20, 2023

STAT1/3 signaling suppresses axon degeneration and neuronal cell death through regulation of NAD+-biosynthetic and consuming enzymes.

STAT1/3 signaling suppresses axon degeneration and neuronal cell death through regulation of NAD+-biosynthetic and consuming enzymes.

Journal: Cellular signalling

Published: January 20, 2023

LOXL1 and LOXL4 are novel target genes of the Zn2+-bound form of ZEB1 and play a crucial role in the acceleration of invasive events in triple-negative breast cancer cells.

LOXL1 and LOXL4 are novel target genes of the Zn2+-bound form of ZEB1 and play a crucial role in the acceleration of invasive events in triple-negative breast cancer cells.

Journal: Frontiers in oncology

Published: January 12, 2023

Lysyl oxidase-like 4 exerts an atypical role in breast cancer progression that is dependent on the enzymatic activity that targets the cell-surface annexin A2.

Lysyl oxidase-like 4 exerts an atypical role in breast cancer progression that is dependent on the enzymatic activity that targets the cell-surface annexin A2.

Journal: Frontiers in oncology

Published: January 12, 2023

Showing 1-12 of 46

Last Updated: 09/26/2026