Hitoshi Okada

Osaka, 5778502, Japan

Last Updated: 09/26/2026

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Found 17 publications

Correction: Deoxycytidine kinase inactivation enhances gemcitabine resistance and sensitizes mitochondrial metabolism interference in pancreatic cancer.

Correction: Deoxycytidine kinase inactivation enhances gemcitabine resistance and sensitizes mitochondrial metabolism interference in pancreatic cancer.

Journal: Cell death & disease
Published: May 31, 2024

Vgll2 as an integrative regulator of mitochondrial function and contractility specific to skeletal muscle.

Vgll2 as an integrative regulator of mitochondrial function and contractility specific to skeletal muscle.

Journal: Journal of cellular physiology
Published: May 13, 2024

Correction: Histone Demethylase JMJD2B Functions as a Co-Factor of Estrogen Receptor in Breast Cancer Proliferation and Mammary Gland Development.

Correction: Histone Demethylase JMJD2B Functions as a Co-Factor of Estrogen Receptor in Breast Cancer Proliferation and Mammary Gland Development.

Journal: PloS one
Published: May 09, 2024

Deoxycytidine kinase inactivation enhances gemcitabine resistance and sensitizes mitochondrial metabolism interference in pancreatic cancer.

Deoxycytidine kinase inactivation enhances gemcitabine resistance and sensitizes mitochondrial metabolism interference in pancreatic cancer.

Journal: Cell death & disease
Published: October 10, 2023

ACA-28, an ERK MAPK Signaling Modulator, Exerts Anticancer Activity through ROS Induction in Melanoma and Pancreatic Cancer Cells.

ACA-28, an ERK MAPK Signaling Modulator, Exerts Anticancer Activity through ROS Induction in Melanoma and Pancreatic Cancer Cells.

Journal: Oxidative medicine and cellular longevity
Published: October 05, 2023

MYC/Glutamine Dependency Is a Therapeutic Vulnerability in Pancreatic Cancer with Deoxycytidine Kinase Inactivation-Induced Gemcitabine Resistance.

MYC/Glutamine Dependency Is a Therapeutic Vulnerability in Pancreatic Cancer with Deoxycytidine Kinase Inactivation-Induced Gemcitabine Resistance.

Journal: Molecular cancer research : MCR
Published: July 13, 2022

KDM4B promotes acute myeloid leukemia associated with AML1-ETO by regulating chromatin accessibility.

KDM4B promotes acute myeloid leukemia associated with AML1-ETO by regulating chromatin accessibility.

Journal: FASEB bioAdvances
Published: February 24, 2021

Copper Promotes Tumorigenesis by Activating the PDK1-AKT Oncogenic Pathway in a Copper Transporter 1 Dependent Manner.

Copper Promotes Tumorigenesis by Activating the PDK1-AKT Oncogenic Pathway in a Copper Transporter 1 Dependent Manner.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)
Published: November 08, 2020

The KDM4B-CCAR1-MED1 axis is a critical regulator of osteoclast differentiation and bone homeostasis.

The KDM4B-CCAR1-MED1 axis is a critical regulator of osteoclast differentiation and bone homeostasis.

Journal: Bone research
Published: August 03, 2020

Tim-3 adaptor protein Bat3 is a molecular checkpoint of T cell terminal differentiation and exhaustion.

Tim-3 adaptor protein Bat3 is a molecular checkpoint of T cell terminal differentiation and exhaustion.

Journal: Science advances
Published: June 11, 2020

Deficiency of Vgll2 Gene Alters the Gene Expression Profiling of Skeletal Muscle Subjected to Mechanical Overload.

Deficiency of Vgll2 Gene Alters the Gene Expression Profiling of Skeletal Muscle Subjected to Mechanical Overload.

Journal: Frontiers in sports and active living
Published: July 17, 2019

High Fat Diet Triggers a Reduction in Body Fat Mass in Female Mice Deficient for Utx demethylase.

High Fat Diet Triggers a Reduction in Body Fat Mass in Female Mice Deficient for Utx demethylase.

Journal: Scientific reports
Published: November 16, 2018
Showing 1-12 of 17

Last Updated: 09/26/2026