Hongjie Zhang
State Key Laboratory Of Cardiology And Medical Innovation Center, Shanghai Key Laboratory Of Signaling And Disease Research
Shanghai, 200092, China
Shanghai, 200092, China
Last Updated: 09/26/2026
Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 10 publications
Myosin inhibition enhances cardiomyocyte cell cycle activity through SIRT1-NFAT-mediated H3K9me3 modification.
Journal: European journal of cell biology
Published: February 28, 2025
miR-199a-3p suppresses Vldlr expression to promote cardiomyocyte proliferation.
Journal: Acta biochimica et biophysica Sinica
Published: February 11, 2025
JAK activity regulates mesoderm cell fate by controlling MESP1 expression.
Journal: European journal of cell biology
Published: July 03, 2024
Impaired end joining induces cardiac atrophy in a Hutchinson-Gilford progeria mouse model.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: November 15, 2023
Thrombospondin 1 and Reelin act through Vldlr to regulate cardiac growth and repair.
Journal: Basic research in cardiology
Published: March 21, 2023
PLK inhibitors identified by high content phenotypic screening promote maturation of human PSC-derived cardiomyocytes.
Journal: Biochemical and biophysical research communications
Published: June 10, 2022
AP-1 activation mediates post-natal cardiomyocyte maturation.
Journal: Cardiovascular research
Published: September 17, 2021
Rcor2 Is Required for Somatic Differentiation and Represses Germline Cell Fate.
Journal: Stem cells international
Published: September 14, 2021
Single-cell transcriptomics of cardiac progenitors reveals functional subpopulations and their cooperative crosstalk in cardiac repair.
Journal: Protein & cell
Published: August 23, 2020
miR-199a-3p promotes cardiomyocyte proliferation by inhibiting Cd151 expression.
Journal: Biochemical and biophysical research communications
Published: April 25, 2019
Showing 1-10 of 10
Last Updated: 09/26/2026