Hongli Li

Chongqing Key Laboratory Of Maternal And Fetal Medicine/Joint International Research Laboratory Of Reproduction & Development, No. 1 Youyi Rd, Yuzhong District
Youyi, 400016, China

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 44 publications

Reduced FBXO22 skews human trophoblast fate equilibrium toward syncytialization via polyubiquitinating the CoREST complex.

Reduced FBXO22 skews human trophoblast fate equilibrium toward syncytialization via polyubiquitinating the CoREST complex.

Journal: Nucleic acids research

Published: December 08, 2025

Aberrant interferon-γ signaling disrupts trophoblast invasion via IDO1/AHR signaling and metabolic reprogramming associated with recurrent pregnancy loss.

Aberrant interferon-γ signaling disrupts trophoblast invasion via IDO1/AHR signaling and metabolic reprogramming associated with recurrent pregnancy loss.

Journal: Life sciences

Published: November 12, 2025

Excessive Kynurenine Metabolism Impairs Lysosomal acidification and Triggers mtDNA Release via the AHR/CISH/ATP6V1A Axis in Decidual Macrophages Associated with Unexplained Recurrent Pregnancy Loss.

Excessive Kynurenine Metabolism Impairs Lysosomal acidification and Triggers mtDNA Release via the AHR/CISH/ATP6V1A Axis in Decidual Macrophages Associated with Unexplained Recurrent Pregnancy Loss.

Journal: International journal of biological sciences

Published: July 18, 2025

Dual strategies for neural injury repair: Targeted modulation of reactive astrocytes and functional astrocyte transplantation.

Dual strategies for neural injury repair: Targeted modulation of reactive astrocytes and functional astrocyte transplantation.

Journal: Neural regeneration research

Published: July 09, 2025

Tunneling nanotube-mediated stem cell immunomodulation dysfunction promotes adipose inflammation and insulin resistance in GDM.

Tunneling nanotube-mediated stem cell immunomodulation dysfunction promotes adipose inflammation and insulin resistance in GDM.

Journal: Cell reports

Published: April 28, 2025

Academic competition-based learning cultivates scientific literacy to promote professional competitiveness in medical undergraduates.

Academic competition-based learning cultivates scientific literacy to promote professional competitiveness in medical undergraduates.

Journal: Frontiers in public health

Published: March 10, 2025

Supervised Factor Analysis Transfer: Calibration transfer with noise modeling and response variable integration.

Supervised Factor Analysis Transfer: Calibration transfer with noise modeling and response variable integration.

Journal: Talanta

Published: May 24, 2024

Gut dysbiosis contributes to SCFAs reduction-associated adipose tissue macrophage polarization in gestational diabetes mellitus.

Gut dysbiosis contributes to SCFAs reduction-associated adipose tissue macrophage polarization in gestational diabetes mellitus.

Journal: Life sciences

Published: March 22, 2024

Oligopeptide-strategy of targeting at adipose tissue macrophages using ATS-9R/siCcl2 complex for ameliorating insulin resistance in GDM.

Oligopeptide-strategy of targeting at adipose tissue macrophages using ATS-9R/siCcl2 complex for ameliorating insulin resistance in GDM.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: March 01, 2024

Application of calcium overload-based ion interference therapy in tumor treatment: strategies, outcomes, and prospects.

Application of calcium overload-based ion interference therapy in tumor treatment: strategies, outcomes, and prospects.

Journal: Frontiers in pharmacology

Published: December 08, 2023

Alternative polyadenylation regulates the translation of metabolic and inflammation-related proteins in adipose tissue of gestational diabetes mellitus.

Alternative polyadenylation regulates the translation of metabolic and inflammation-related proteins in adipose tissue of gestational diabetes mellitus.

Journal: Computational and structural biotechnology journal

Published: November 23, 2023

Osteopontin deficiency promotes cartilaginous endplate degeneration by enhancing the NF-κB signaling to recruit macrophages and activate the NLRP3 inflammasome.

Osteopontin deficiency promotes cartilaginous endplate degeneration by enhancing the NF-κB signaling to recruit macrophages and activate the NLRP3 inflammasome.

Journal: Bone research

Published: September 27, 2023

Showing 1-12 of 44

Last Updated: 09/26/2026