Hongxiang Lu
Nanjing, China
Last Updated: 09/26/2026
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Found 16 publications
3C suppresses PINK1-mediated mitophagy and contributes to coxsackievirus B3 replication.
Journal: Virulence
Published: April 26, 2026
sST2: A Bridge Between Sirt1/p53/p21 Signal-Induced Senescence and TGF-β1/Smad2/3 Regulation of Cardiac Fibrosis in Mouse Viral Myocarditis.
Journal: Inflammation
Published: December 06, 2022
Retraction Note: HMGB1 Facilitated Macrophage Reprogramming towards a Proinflammatory M1-like Phenotype in Experimental Autoimmune Myocarditis Development.
Journal: Scientific reports
Published: April 09, 2022
Fibroblast transdifferentiation promotes conversion of M1 macrophages and replenishment of cardiac resident macrophages following cardiac injury in mice.
Journal: European journal of immunology
Published: September 22, 2019
PGE2 ameliorated viral myocarditis development and promoted IL-10-producing regulatory B cell expansion via MAPKs/AKT-AP1 axis or AhR signaling.
Journal: Cellular immunology
Published: August 21, 2019
Dual faced HMGB1 plays multiple roles in cardiomyocyte senescence and cardiac inflammatory injury.
Journal: Cytokine & growth factor reviews
Published: February 21, 2019
Reg3β from cardiomyocytes regulated macrophage migration, proliferation and functional skewing in experimental autoimmune myocarditis.
Journal: American journal of clinical and experimental immunology
Published: March 08, 2018
Angiotensin II enhances the acetylation and release of HMGB1 in RAW264.7 macrophage.
Journal: Cell biology international
Published: March 07, 2018
HMGB1 silencing in macrophages prevented their functional skewing and ameliorated EAM development: Nuclear HMGB1 may be a checkpoint molecule of macrophage reprogramming.
Journal: International immunopharmacology
Published: October 13, 2017
ANG II facilitated CD11+ Ly6Chi cells reprogramming into M1-like macrophage through Erk1/2 or p38-Stat3 pathway and involved in EAM.
Journal: Journal of leukocyte biology
Published: June 29, 2017
Last Updated: 09/26/2026