Hui-ling Jiang

Changsha, China

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 10 publications

CGRP is essential for protection against alveolar epithelial cell necroptosis by activating the AMPK/L-OPA1 signaling pathway during acute lung injury.

CGRP is essential for protection against alveolar epithelial cell necroptosis by activating the AMPK/L-OPA1 signaling pathway during acute lung injury.

Journal: Journal of cellular physiology

Published: September 10, 2023

Mitochondrial citrate accumulation drives alveolar epithelial cell necroptosis in lipopolysaccharide-induced acute lung injury.

Mitochondrial citrate accumulation drives alveolar epithelial cell necroptosis in lipopolysaccharide-induced acute lung injury.

Journal: Experimental & molecular medicine

Published: July 30, 2022

L-OPA1 deficiency aggravates necroptosis of alveolar epithelial cells through impairing mitochondrial function during acute lung injury in mice.

L-OPA1 deficiency aggravates necroptosis of alveolar epithelial cells through impairing mitochondrial function during acute lung injury in mice.

Journal: Journal of cellular physiology

Published: January 20, 2022

Fn14 exacerbates acute lung injury by activating the NLRP3 inflammasome in mice.

Fn14 exacerbates acute lung injury by activating the NLRP3 inflammasome in mice.

Journal: Molecular medicine (Cambridge, Mass.)

Published: January 03, 2022

COX-2/sEH Dual Inhibitor PTUPB Attenuates Epithelial-Mesenchymal Transformation of Alveolar Epithelial Cells via Nrf2-Mediated Inhibition of TGF-β1/Smad Signaling.

COX-2/sEH Dual Inhibitor PTUPB Attenuates Epithelial-Mesenchymal Transformation of Alveolar Epithelial Cells via Nrf2-Mediated Inhibition of TGF-β1/Smad Signaling.

Journal: Oxidative medicine and cellular longevity

Published: December 18, 2021

Extracellular citrate serves as a DAMP to activate macrophages and promote LPS-induced lung injury in mice.

Extracellular citrate serves as a DAMP to activate macrophages and promote LPS-induced lung injury in mice.

Journal: International immunopharmacology

Published: August 09, 2021

PTUPB ameliorates high-fat diet-induced non-alcoholic fatty liver disease via inhibiting NLRP3 inflammasome activation in mice.

PTUPB ameliorates high-fat diet-induced non-alcoholic fatty liver disease via inhibiting NLRP3 inflammasome activation in mice.

Journal: Biochemical and biophysical research communications

Published: December 04, 2019

A COX-2/sEH dual inhibitor PTUPB ameliorates cecal ligation and puncture-induced sepsis in mice via anti-inflammation and anti-oxidative stress.

A COX-2/sEH dual inhibitor PTUPB ameliorates cecal ligation and puncture-induced sepsis in mice via anti-inflammation and anti-oxidative stress.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: September 24, 2019

COX-2/sEH dual inhibitor PTUPB alleviates bleomycin-induced pulmonary fibrosis in mice via inhibiting senescence.

COX-2/sEH dual inhibitor PTUPB alleviates bleomycin-induced pulmonary fibrosis in mice via inhibiting senescence.

Journal: The FEBS journal

Published: June 21, 2019

Exogenous angiotensin (1-7) directly inhibits epithelial-mesenchymal transformation induced by transforming growth factor-β1 in alveolar epithelial cells.

Exogenous angiotensin (1-7) directly inhibits epithelial-mesenchymal transformation induced by transforming growth factor-β1 in alveolar epithelial cells.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: March 20, 2019

Showing 1-10 of 10

Last Updated: 09/26/2026