Huiyao Cai

No. 34 North Zhongshan Road
Quanzhou, 362000, China

Last Updated: 09/26/2026

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Found 14 publications

Follicular helper T cells in Graves' disease: pathogenic mechanisms and therapeutic implications.

Follicular helper T cells in Graves' disease: pathogenic mechanisms and therapeutic implications.

Journal: American journal of physiology. Endocrinology and metabolism

Published: May 05, 2025

Circular RNA circPHF16 enhances IL-17A expression and secretion by sequestering miR-378a-3p to activate the IL6ST axis in Graves' disease.

Circular RNA circPHF16 enhances IL-17A expression and secretion by sequestering miR-378a-3p to activate the IL6ST axis in Graves' disease.

Journal: Cytokine

Published: March 27, 2024

PTPN22 through the regulation of Th17/Treg balance acts as a potential target for the treatment of Graves' disease.

PTPN22 through the regulation of Th17/Treg balance acts as a potential target for the treatment of Graves' disease.

Journal: Tissue & cell

Published: February 23, 2024

T-cell exhaustion-related genes in Graves' disease: a comprehensive genome mapping analysis.

T-cell exhaustion-related genes in Graves' disease: a comprehensive genome mapping analysis.

Journal: Frontiers in endocrinology

Published: January 03, 2024

TSHR-based chimeric antigen receptor T cell specifically deplete auto-reactive B lymphocytes for treatment of autoimmune thyroid disease.

TSHR-based chimeric antigen receptor T cell specifically deplete auto-reactive B lymphocytes for treatment of autoimmune thyroid disease.

Journal: International immunopharmacology

Published: May 06, 2023

Circular RNA HIPK3 contributes to hyperglycemia and insulin homeostasis by sponging miR-192-5p and upregulating transcription factor forkhead box O1.

Circular RNA HIPK3 contributes to hyperglycemia and insulin homeostasis by sponging miR-192-5p and upregulating transcription factor forkhead box O1.

Journal: Endocrine journal

Published: December 26, 2019

Optimal iodine supplementation during antithyroid drug therapy for Graves' disease is associated with lower recurrence rates than iodine restriction.

Optimal iodine supplementation during antithyroid drug therapy for Graves' disease is associated with lower recurrence rates than iodine restriction.

Journal: Clinical endocrinology

Published: July 11, 2017

Iodinated TG in Thyroid Follicles Regulate TSH/TSHR Signaling for NIS Expression.

Iodinated TG in Thyroid Follicles Regulate TSH/TSHR Signaling for NIS Expression.

Journal: Biological trace element research

Published: February 13, 2017

Iodinated TG in Thyroid Follicular Lumen Regulates TTF-1 and PAX8 Expression via TSH/TSHR Signaling Pathway.

Iodinated TG in Thyroid Follicular Lumen Regulates TTF-1 and PAX8 Expression via TSH/TSHR Signaling Pathway.

Journal: Journal of cellular biochemistry

Published: February 08, 2017

Low Iodine in the Follicular Lumen Caused by Cytoplasm Mis-localization of Sodium Iodide Symporter may Induce Nodular Goiter.

Low Iodine in the Follicular Lumen Caused by Cytoplasm Mis-localization of Sodium Iodide Symporter may Induce Nodular Goiter.

Journal: Biological trace element research

Published: December 03, 2016

Glycolysis-associated enzymes existing in the follicular lumen of the thyroid may interfere with energy metabolism.

Glycolysis-associated enzymes existing in the follicular lumen of the thyroid may interfere with energy metabolism.

Journal: Biomedical reports

Published: May 10, 2016

Upregulation of TSHR, TTF-1, and PAX8 in Nodular Goiter Is Associated with Iodine Deficiency in the Follicular Lumen.

Upregulation of TSHR, TTF-1, and PAX8 in Nodular Goiter Is Associated with Iodine Deficiency in the Follicular Lumen.

Journal: International journal of endocrinology

Published: April 06, 2016

Showing 1-12 of 14

Last Updated: 09/26/2026