Hwan-jin Hwang

Seoul, Republic of Korea

Last Updated: 09/26/2026

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Found 22 publications

GPR40-full agonist AM1638 alleviates palmitate-induced oxidative damage in H9c2 cells via an AMPK-dependent pathway.

GPR40-full agonist AM1638 alleviates palmitate-induced oxidative damage in H9c2 cells via an AMPK-dependent pathway.

Journal: BMB reports

Published: March 25, 2024

HD6277 Suppresses Muscle Atrophy by Promoting Myogenic Factors and Inhibiting Proteolysis in Aged Mice.

HD6277 Suppresses Muscle Atrophy by Promoting Myogenic Factors and Inhibiting Proteolysis in Aged Mice.

Journal: Journal of cachexia, sarcopenia and muscle

Published: March 11, 2024

AM1638, a GPR40-Full Agonist, Inhibited Palmitate- Induced ROS Production and Endoplasmic Reticulum Stress, Enhancing HUVEC Viability in an NRF2-Dependent Manner.

AM1638, a GPR40-Full Agonist, Inhibited Palmitate- Induced ROS Production and Endoplasmic Reticulum Stress, Enhancing HUVEC Viability in an NRF2-Dependent Manner.

Journal: Endocrinology and metabolism (Seoul, Korea)

Published: July 04, 2023

Sestrin2 Regulates Beneficial β3-Adrenergic Receptor-Mediated Effects Observed in Inguinal White Adipose Tissue and Soleus Muscle.

Sestrin2 Regulates Beneficial β3-Adrenergic Receptor-Mediated Effects Observed in Inguinal White Adipose Tissue and Soleus Muscle.

Journal: Endocrinology and metabolism (Seoul, Korea)

Published: February 19, 2022

GPR40 Agonism Modulates Inflammatory Reactions in Vascular Endothelial Cells.

GPR40 Agonism Modulates Inflammatory Reactions in Vascular Endothelial Cells.

Journal: Diabetes & metabolism journal

Published: May 11, 2021

Ginsenoside Mc1 improves liver steatosis and insulin resistance by attenuating ER stress.

Ginsenoside Mc1 improves liver steatosis and insulin resistance by attenuating ER stress.

Journal: Journal of ethnopharmacology

Published: December 28, 2019

Association of serum FAM19A5 with metabolic and vascular risk factors in human subjects with or without type 2 diabetes.

Association of serum FAM19A5 with metabolic and vascular risk factors in human subjects with or without type 2 diabetes.

Journal: Diabetes & vascular disease research

Published: July 09, 2019

Ginsenoside compound-Mc1 attenuates oxidative stress and apoptosis in cardiomyocytes through an AMP-activated protein kinase-dependent mechanism.

Ginsenoside compound-Mc1 attenuates oxidative stress and apoptosis in cardiomyocytes through an AMP-activated protein kinase-dependent mechanism.

Journal: Journal of ginseng research

Published: December 07, 2018

Erratum to "Knockdown of Sestrin2 Increases Lipopolysaccharide-Induced Oxidative Stress, Apoptosis, and Fibrotic Reactions in H9c2 Cells and Heart Tissues of Mice via an AMPK-Dependent Mechanism".

Erratum to "Knockdown of Sestrin2 Increases Lipopolysaccharide-Induced Oxidative Stress, Apoptosis, and Fibrotic Reactions in H9c2 Cells and Heart Tissues of Mice via an AMPK-Dependent Mechanism".

Journal: Mediators of inflammation

Published: December 05, 2018

Protectin DX prevents H2O2-mediated oxidative stress in vascular endothelial cells via an AMPK-dependent mechanism.

Protectin DX prevents H2O2-mediated oxidative stress in vascular endothelial cells via an AMPK-dependent mechanism.

Journal: Cellular signalling

Published: June 20, 2018

Association of serum Sestrin2 level with metabolic risk factors in newly diagnosed drug-naïve type 2 diabetes.

Association of serum Sestrin2 level with metabolic risk factors in newly diagnosed drug-naïve type 2 diabetes.

Journal: Diabetes research and clinical practice

Published: March 01, 2018

Knockdown of Sestrin2 Increases Lipopolysaccharide-Induced Oxidative Stress, Apoptosis, and Fibrotic Reactions in H9c2 Cells and Heart Tissues of Mice via an AMPK-Dependent Mechanism.

Knockdown of Sestrin2 Increases Lipopolysaccharide-Induced Oxidative Stress, Apoptosis, and Fibrotic Reactions in H9c2 Cells and Heart Tissues of Mice via an AMPK-Dependent Mechanism.

Journal: Mediators of inflammation

Published: December 04, 2017

Showing 1-12 of 22

Last Updated: 09/26/2026