Hyejin J. Kong

St. Paul's Hospital
Vancouver, BC, Canada

Last Updated: 09/26/2026

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Found 12 publications

Complex Thrombo-Inflammatory Responses versus Outcomes of Non-COVID-19 Community-Acquired Pneumonia and COVID-19.

Complex Thrombo-Inflammatory Responses versus Outcomes of Non-COVID-19 Community-Acquired Pneumonia and COVID-19.

Journal: Journal of innate immunity
Published: February 07, 2024

Modulation of vascular endothelial inflammatory response by proprotein convertase subtilisin-kexin type 9.

Modulation of vascular endothelial inflammatory response by proprotein convertase subtilisin-kexin type 9.

Journal: Atherosclerosis
Published: April 12, 2022

Very Low Density Lipoprotein Receptor Sequesters Lipopolysaccharide Into Adipose Tissue During Sepsis.

Very Low Density Lipoprotein Receptor Sequesters Lipopolysaccharide Into Adipose Tissue During Sepsis.

Journal: Critical care medicine
Published: October 26, 2019

Oligonucleotide-based Preconditioning of DCD Cardiac Donors and Its Impact on Cardiac Viability.

Oligonucleotide-based Preconditioning of DCD Cardiac Donors and Its Impact on Cardiac Viability.

Journal: Transplantation
Published: July 24, 2019

Low Low-Density Lipoprotein Levels Are Associated With, But Do Not Causally Contribute to, Increased Mortality in Sepsis.

Low Low-Density Lipoprotein Levels Are Associated With, But Do Not Causally Contribute to, Increased Mortality in Sepsis.

Journal: Critical care medicine
Published: November 06, 2018

Cholesteryl Ester Transfer Protein Influences High-Density Lipoprotein Levels and Survival in Sepsis.

Cholesteryl Ester Transfer Protein Influences High-Density Lipoprotein Levels and Survival in Sepsis.

Journal: American journal of respiratory and critical care medicine
Published: October 16, 2018

CETP genetic variant rs1800777 (allele A) is associated with abnormally low HDL-C levels and increased risk of AKI during sepsis.

CETP genetic variant rs1800777 (allele A) is associated with abnormally low HDL-C levels and increased risk of AKI during sepsis.

Journal: Scientific reports
Published: July 20, 2018

Is Heparin-Binding Protein Inhibition a Mechanism of Albumin's Efficacy in Human Septic Shock?

Is Heparin-Binding Protein Inhibition a Mechanism of Albumin's Efficacy in Human Septic Shock?

Journal: Critical care medicine
Published: February 07, 2018

Erratum to: Heparin-binding protein is important for vascular leak in sepsis.

Erratum to: Heparin-binding protein is important for vascular leak in sepsis.

Journal: Intensive care medicine experimental
Published: January 12, 2017

Correction: Lipopolysaccharide Is Cleared from the Circulation by Hepatocytes via the Low Density Lipoprotein Receptor.

Correction: Lipopolysaccharide Is Cleared from the Circulation by Hepatocytes via the Low Density Lipoprotein Receptor.

Journal: PloS one
Published: July 27, 2016

Heparin-binding protein is important for vascular leak in sepsis.

Heparin-binding protein is important for vascular leak in sepsis.

Journal: Intensive care medicine experimental
Published: March 17, 2016

Lipopolysaccharide Is Cleared from the Circulation by Hepatocytes via the Low Density Lipoprotein Receptor.

Lipopolysaccharide Is Cleared from the Circulation by Hepatocytes via the Low Density Lipoprotein Receptor.

Journal: PloS one
Published: March 05, 2016
Showing 1-12 of 12

Last Updated: 09/26/2026