Hyunjung Ha

Cheongju-si, Republic of Korea

Last Updated: 09/26/2026

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Found 13 publications

R-PTP-κ Inhibits Contact-Dependent Cell Growth by Suppressing E2F Activity.

R-PTP-κ Inhibits Contact-Dependent Cell Growth by Suppressing E2F Activity.

Journal: Biomedicines

Published: October 19, 2022

DAXX ameliorates metabolic dysfunction in mice with diet-induced obesity by activating the AMP-activated protein kinase-related kinase MPK38/MELK.

DAXX ameliorates metabolic dysfunction in mice with diet-induced obesity by activating the AMP-activated protein kinase-related kinase MPK38/MELK.

Journal: Biochemical and biophysical research communications

Published: July 12, 2021

Ablation of AMPK-Related Kinase MPK38/MELK Leads to Male-Specific Obesity in Aged Mature Adult Mice.

Ablation of AMPK-Related Kinase MPK38/MELK Leads to Male-Specific Obesity in Aged Mature Adult Mice.

Journal: Diabetes

Published: April 29, 2020

Thr55 phosphorylation of p21 by MPK38/MELK ameliorates defects in glucose, lipid, and energy metabolism in diet-induced obese mice.

Thr55 phosphorylation of p21 by MPK38/MELK ameliorates defects in glucose, lipid, and energy metabolism in diet-induced obese mice.

Journal: Cell death & disease

Published: January 25, 2019

Dual Roles of Serine-Threonine Kinase Receptor-Associated Protein (STRAP) in Redox-Sensitive Signaling Pathways Related to Cancer Development.

Dual Roles of Serine-Threonine Kinase Receptor-Associated Protein (STRAP) in Redox-Sensitive Signaling Pathways Related to Cancer Development.

Journal: Oxidative medicine and cellular longevity

Published: December 17, 2017

Smad proteins differentially regulate obesity-induced glucose and lipid abnormalities and inflammation via class-specific control of AMPK-related kinase MPK38/MELK activity.

Smad proteins differentially regulate obesity-induced glucose and lipid abnormalities and inflammation via class-specific control of AMPK-related kinase MPK38/MELK activity.

Journal: Cell death & disease

Published: October 03, 2017

Macrophage migration inhibitory factor interacts with thioredoxin-interacting protein and induces NF-κB activity.

Macrophage migration inhibitory factor interacts with thioredoxin-interacting protein and induces NF-κB activity.

Journal: Cellular signalling

Published: January 11, 2017

Zinc finger protein ZPR9 functions as an activator of AMPK-related serine/threonine kinase MPK38/MELK involved in ASK1/TGF-β/p53 signaling pathways.

Zinc finger protein ZPR9 functions as an activator of AMPK-related serine/threonine kinase MPK38/MELK involved in ASK1/TGF-β/p53 signaling pathways.

Journal: Scientific reports

Published: August 26, 2016

Coordinate Activation of Redox-Dependent ASK1/TGF-β Signaling by a Multiprotein Complex (MPK38, ASK1, SMADs, ZPR9, and TRX) Improves Glucose and Lipid Metabolism in Mice.

Coordinate Activation of Redox-Dependent ASK1/TGF-β Signaling by a Multiprotein Complex (MPK38, ASK1, SMADs, ZPR9, and TRX) Improves Glucose and Lipid Metabolism in Mice.

Journal: Antioxidants & redox signaling

Published: October 01, 2015

Thioredoxin inhibits MPK38-induced ASK1, TGF-β, and p53 function in a phosphorylation-dependent manner.

Thioredoxin inhibits MPK38-induced ASK1, TGF-β, and p53 function in a phosphorylation-dependent manner.

Journal: Free radical biology & medicine

Published: February 08, 2013

PDK1 protein phosphorylation at Thr354 by murine protein serine-threonine kinase 38 contributes to negative regulation of PDK1 protein activity.

PDK1 protein phosphorylation at Thr354 by murine protein serine-threonine kinase 38 contributes to negative regulation of PDK1 protein activity.

Journal: The Journal of biological chemistry

Published: May 01, 2012

Murine protein serine-threonine kinase 38 activates p53 function through Ser15 phosphorylation.

Murine protein serine-threonine kinase 38 activates p53 function through Ser15 phosphorylation.

Journal: The Journal of biological chemistry

Published: April 26, 2012

Showing 1-12 of 13

Last Updated: 09/26/2026