Ilana Shapira

Tel Aviv, TA 69978, Israel

Last Updated: 09/26/2026

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Found 8 publications

Deep brain stimulation of thalamic nucleus reuniens promotes neuronal and cognitive resilience in an Alzheimer's disease mouse model.

Deep brain stimulation of thalamic nucleus reuniens promotes neuronal and cognitive resilience in an Alzheimer's disease mouse model.

Journal: Nature communications

Published: October 15, 2022

NMDA receptors regulate the firing rate set point of hippocampal circuits without altering single-cell dynamics.

NMDA receptors regulate the firing rate set point of hippocampal circuits without altering single-cell dynamics.

Journal: Neuron

Published: October 11, 2022

IGF-1 receptor regulates upward firing rate homeostasis via the mitochondrial calcium uniporter.

IGF-1 receptor regulates upward firing rate homeostasis via the mitochondrial calcium uniporter.

Journal: Proceedings of the National Academy of Sciences of the United States of America

Published: August 09, 2022

Disrupted neural correlates of anesthesia and sleep reveal early circuit dysfunctions in Alzheimer models.

Disrupted neural correlates of anesthesia and sleep reveal early circuit dysfunctions in Alzheimer models.

Journal: Cell reports

Published: November 01, 2020

Mitochondrial Regulation of the Hippocampal Firing Rate Set Point and Seizure Susceptibility.

Mitochondrial Regulation of the Hippocampal Firing Rate Set Point and Seizure Susceptibility.

Journal: Neuron

Published: July 03, 2018

GABAB receptor deficiency causes failure of neuronal homeostasis in hippocampal networks.

GABAB receptor deficiency causes failure of neuronal homeostasis in hippocampal networks.

Journal: Proceedings of the National Academy of Sciences of the United States of America

Published: June 10, 2015

IGF-1 Receptor Differentially Regulates Spontaneous and Evoked Transmission via Mitochondria at Hippocampal Synapses.

IGF-1 Receptor Differentially Regulates Spontaneous and Evoked Transmission via Mitochondria at Hippocampal Synapses.

Journal: Neuron

Published: December 13, 2014

APP homodimers transduce an amyloid-β-mediated increase in release probability at excitatory synapses.

APP homodimers transduce an amyloid-β-mediated increase in release probability at excitatory synapses.

Journal: Cell reports

Published: August 28, 2012

Showing 1-8 of 8

Last Updated: 09/26/2026