Ilaria Zuliani

Rome, 00185, Italy

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 9 publications

Biliverdin reductase-A protein levels are reduced in type 2 diabetes and are associated with poor glycometabolic control.

Biliverdin reductase-A protein levels are reduced in type 2 diabetes and are associated with poor glycometabolic control.

Journal: Life sciences

Published: July 14, 2021

High-Fat Diet Leads to Reduced Protein O-GlcNAcylation and Mitochondrial Defects Promoting the Development of Alzheimer's Disease Signatures.

High-Fat Diet Leads to Reduced Protein O-GlcNAcylation and Mitochondrial Defects Promoting the Development of Alzheimer's Disease Signatures.

Journal: International journal of molecular sciences

Published: January 29, 2021

The Dysregulation of OGT/OGA Cycle Mediates Tau and APP Neuropathology in Down Syndrome.

The Dysregulation of OGT/OGA Cycle Mediates Tau and APP Neuropathology in Down Syndrome.

Journal: Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics

Published: November 18, 2020

BVR-A Deficiency Leads to Autophagy Impairment through the Dysregulation of AMPK/mTOR Axis in the Brain-Implications for Neurodegeneration.

BVR-A Deficiency Leads to Autophagy Impairment through the Dysregulation of AMPK/mTOR Axis in the Brain-Implications for Neurodegeneration.

Journal: Antioxidants (Basel, Switzerland)

Published: June 30, 2020

The Anti-Diabetic Drug Metformin Rescues Aberrant Mitochondrial Activity and Restrains Oxidative Stress in a Female Mouse Model of Rett Syndrome.

The Anti-Diabetic Drug Metformin Rescues Aberrant Mitochondrial Activity and Restrains Oxidative Stress in a Female Mouse Model of Rett Syndrome.

Journal: Journal of clinical medicine

Published: April 28, 2020

Chronic PERK induction promotes Alzheimer-like neuropathology in Down syndrome: Insights for therapeutic intervention.

Chronic PERK induction promotes Alzheimer-like neuropathology in Down syndrome: Insights for therapeutic intervention.

Journal: Progress in neurobiology

Published: March 18, 2020

Shining a light on defective autophagy by proteomics approaches: implications for neurodegenerative illnesses.

Shining a light on defective autophagy by proteomics approaches: implications for neurodegenerative illnesses.

Journal: Expert review of proteomics

Published: November 12, 2019

Restoration of aberrant mTOR signaling by intranasal rapamycin reduces oxidative damage: Focus on HNE-modified proteins in a mouse model of down syndrome.

Restoration of aberrant mTOR signaling by intranasal rapamycin reduces oxidative damage: Focus on HNE-modified proteins in a mouse model of down syndrome.

Journal: Redox biology

Published: October 29, 2018

Intranasal rapamycin ameliorates Alzheimer-like cognitive decline in a mouse model of Down syndrome.

Intranasal rapamycin ameliorates Alzheimer-like cognitive decline in a mouse model of Down syndrome.

Journal: Translational neurodegeneration

Published: July 16, 2018

Showing 1-9 of 9

Last Updated: 09/26/2026