Iman Gohari-Mehdizadeh

Guelph, ON, Canada

Last Updated: 09/26/2026

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Found 26 publications

BrnQ Branched-Chain Amino Acid Transporters Influence Toxin Production by, but Not Growth of, Clostridium perfringens Type A Strain ATCC3624.

BrnQ Branched-Chain Amino Acid Transporters Influence Toxin Production by, but Not Growth of, Clostridium perfringens Type A Strain ATCC3624.

Journal: Toxins

Published: December 18, 2024

Heat resistance differences are common between both vegetative cells and spores of Clostridium perfringens type F isolates carrying a chromosomal vs plasmid-borne enterotoxin gene.

Heat resistance differences are common between both vegetative cells and spores of Clostridium perfringens type F isolates carrying a chromosomal vs plasmid-borne enterotoxin gene.

Journal: Applied and environmental microbiology

Published: September 18, 2024

The biology and pathogenicity of Clostridium perfringens type F: a common human enteropathogen with a new(ish) name.

The biology and pathogenicity of Clostridium perfringens type F: a common human enteropathogen with a new(ish) name.

Journal: Microbiology and molecular biology reviews : MMBR

Published: June 12, 2024

The impact of orphan histidine kinases and phosphotransfer proteins on the regulation of clostridial sporulation initiation.

The impact of orphan histidine kinases and phosphotransfer proteins on the regulation of clostridial sporulation initiation.

Journal: mBio

Published: March 13, 2024

Overexpressing the cpr1953 Orphan Histidine Kinase Gene in the Absence of cpr1954 Orphan Histidine Kinase Gene Expression, or Vice Versa, Is Sufficient to Obtain Significant Sporulation and Strong Production of Clostridium perfringens Enterotoxin or Spo0A by Clostridium perfringens Type F Strain SM101.

Overexpressing the cpr1953 Orphan Histidine Kinase Gene in the Absence of cpr1954 Orphan Histidine Kinase Gene Expression, or Vice Versa, Is Sufficient to Obtain Significant Sporulation and Strong Production of Clostridium perfringens Enterotoxin or Spo0A by Clostridium perfringens Type F Strain SM101.

Journal: Toxins

Published: March 13, 2024

Identification of orphan histidine kinases that impact sporulation and enterotoxin production by Clostridium perfringens type F strain SM101 in a pathophysiologically-relevant ex vivo mouse intestinal contents model.

Identification of orphan histidine kinases that impact sporulation and enterotoxin production by Clostridium perfringens type F strain SM101 in a pathophysiologically-relevant ex vivo mouse intestinal contents model.

Journal: PLoS pathogens

Published: November 22, 2022

Reevaluation of whether a Functional Agr-like Quorum-Sensing System Is Necessary for Production of Wild-Type Levels of Epsilon-Toxin by Clostridium perfringens Type D Strains.

Reevaluation of whether a Functional Agr-like Quorum-Sensing System Is Necessary for Production of Wild-Type Levels of Epsilon-Toxin by Clostridium perfringens Type D Strains.

Journal: mBio

Published: March 23, 2022

Identifying the Basis for VirS/VirR Two-Component Regulatory System Control of Clostridium perfringens Beta-Toxin Production.

Identifying the Basis for VirS/VirR Two-Component Regulatory System Control of Clostridium perfringens Beta-Toxin Production.

Journal: Journal of bacteriology

Published: July 06, 2021

Pathogenicity and virulence of Clostridium perfringens.

Pathogenicity and virulence of Clostridium perfringens.

Journal: Virulence

Published: April 12, 2021

Clostridium perfringens Produces an Adhesive Pilus Required for the Pathogenesis of Necrotic Enteritis in Poultry.

Clostridium perfringens Produces an Adhesive Pilus Required for the Pathogenesis of Necrotic Enteritis in Poultry.

Journal: Journal of bacteriology

Published: October 19, 2020

NetF-producing Clostridium perfringens and its associated diseases in dogs and foals.

NetF-producing Clostridium perfringens and its associated diseases in dogs and foals.

Journal: Journal of veterinary diagnostic investigation : official publication of the American Association of Veterinary Laboratory Diagnosticians, Inc

Published: February 22, 2020

RIP1, RIP3, and MLKL Contribute to Cell Death Caused by Clostridium perfringens Enterotoxin.

RIP1, RIP3, and MLKL Contribute to Cell Death Caused by Clostridium perfringens Enterotoxin.

Journal: mBio

Published: December 19, 2019

Showing 1-12 of 26

Last Updated: 09/26/2026