Inger Lauritzen

Valbonne, France

Last Updated: 09/26/2026

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Found 23 publications

PRKN/parkin-mediated control of SNCA (synuclein alpha) and chaperone-mediated autophagy are defective in cellular, mice models and Parkinson disease-affected brains.

PRKN/parkin-mediated control of SNCA (synuclein alpha) and chaperone-mediated autophagy are defective in cellular, mice models and Parkinson disease-affected brains.

Journal: Autophagy

Published: May 05, 2026

Functional relationships linking C99/APP-βCTF dimerization, proteostasis disruption, and organelle dysfunction.

Functional relationships linking C99/APP-βCTF dimerization, proteostasis disruption, and organelle dysfunction.

Journal: Cell communication and signaling : CCS

Published: November 14, 2025

Presenilins as hub proteins controlling the endocytic and autophagic pathways and small extracellular vesicle secretion.

Presenilins as hub proteins controlling the endocytic and autophagic pathways and small extracellular vesicle secretion.

Journal: Journal of extracellular vesicles

Published: February 05, 2024

Correction to: Intraneuronal aggregation of the β‑CTF fragment of APP (C99) induces Aβ‑independent lysosomal-autophagic pathology.

Correction to: Intraneuronal aggregation of the β‑CTF fragment of APP (C99) induces Aβ‑independent lysosomal-autophagic pathology.

Journal: Acta neuropathologica

Published: August 04, 2023

The η-secretase-derived APP fragment ηCTF is localized in Golgi, endosomes and extracellular vesicles and contributes to Aβ production.

The η-secretase-derived APP fragment ηCTF is localized in Golgi, endosomes and extracellular vesicles and contributes to Aβ production.

Journal: Cellular and molecular life sciences : CMLS

Published: November 18, 2022

Transcription- and phosphorylation-dependent control of a functional interplay between XBP1s and PINK1 governs mitophagy and potentially impacts Parkinson disease pathophysiology.

Transcription- and phosphorylation-dependent control of a functional interplay between XBP1s and PINK1 governs mitophagy and potentially impacts Parkinson disease pathophysiology.

Journal: Autophagy

Published: May 25, 2021

Is γ-secretase a beneficial inactivating enzyme of the toxic APP C-terminal fragment C99?

Is γ-secretase a beneficial inactivating enzyme of the toxic APP C-terminal fragment C99?

Journal: The Journal of biological chemistry

Published: October 09, 2020

Accumulation of amyloid precursor protein C-terminal fragments triggers mitochondrial structure, function, and mitophagy defects in Alzheimer's disease models and human brains.

Accumulation of amyloid precursor protein C-terminal fragments triggers mitochondrial structure, function, and mitophagy defects in Alzheimer's disease models and human brains.

Journal: Acta neuropathologica

Published: June 09, 2020

The Transcription Factor EB Reduces the Intraneuronal Accumulation of the Beta-Secretase-Derived APP Fragment C99 in Cellular and Mouse Alzheimer’s Disease Models.

The Transcription Factor EB Reduces the Intraneuronal Accumulation of the Beta-Secretase-Derived APP Fragment C99 in Cellular and Mouse Alzheimer’s Disease Models.

Journal: Cells

Published: April 14, 2020

Targeting γ-secretase triggers the selective enrichment of oligomeric APP-CTFs in brain extracellular vesicles from Alzheimer cell and mouse models.

Targeting γ-secretase triggers the selective enrichment of oligomeric APP-CTFs in brain extracellular vesicles from Alzheimer cell and mouse models.

Journal: Translational neurodegeneration

Published: August 10, 2019

Does Intraneuronal Accumulation of Carboxyl-terminal Fragments of the Amyloid Precursor Protein Trigger Early Neurotoxicity in Alzheimer's Disease?

Does Intraneuronal Accumulation of Carboxyl-terminal Fragments of the Amyloid Precursor Protein Trigger Early Neurotoxicity in Alzheimer's Disease?

Journal: Current Alzheimer research

Published: October 26, 2018

Chronic fornix deep brain stimulation in a transgenic Alzheimer's rat model reduces amyloid burden, inflammation, and neuronal loss.

Chronic fornix deep brain stimulation in a transgenic Alzheimer's rat model reduces amyloid burden, inflammation, and neuronal loss.

Journal: Brain structure & function

Published: July 18, 2018

Showing 1-12 of 23

Last Updated: 09/26/2026