Isabel Zvibel

Tel Aviv, TA, Israel

Last Updated: 09/26/2026

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Found 16 publications

S100A8/S100A9 impairs energy expenditure and whole body metabolism.

S100A8/S100A9 impairs energy expenditure and whole body metabolism.

Journal: American journal of physiology. Endocrinology and metabolism

Published: July 15, 2025

Sortilin in Biliary Epithelial Cells Promotes Ductular Reaction and Fibrosis during Cholestatic Injury.

Sortilin in Biliary Epithelial Cells Promotes Ductular Reaction and Fibrosis during Cholestatic Injury.

Journal: The American journal of pathology

Published: September 11, 2023

NLRP3 Deficiency in Nonimmune Cells Averts Obesity-Induced Fatty Liver Disease.

NLRP3 Deficiency in Nonimmune Cells Averts Obesity-Induced Fatty Liver Disease.

Journal: Laboratory investigation; a journal of technical methods and pathology

Published: August 03, 2023

Repetitive amiodarone administration causes liver damage via adipose tissue ER stress-dependent lipolysis, leading to hepatotoxic free fatty acid accumulation.

Repetitive amiodarone administration causes liver damage via adipose tissue ER stress-dependent lipolysis, leading to hepatotoxic free fatty acid accumulation.

Journal: American journal of physiology. Gastrointestinal and liver physiology

Published: July 14, 2021

GIPR Signaling in Immune Cells Maintains Metabolically Beneficial Type 2 Immune Responses in the White Fat From Obese Mice.

GIPR Signaling in Immune Cells Maintains Metabolically Beneficial Type 2 Immune Responses in the White Fat From Obese Mice.

Journal: Frontiers in immunology

Published: December 17, 2020

GIP regulates inflammation and body weight by restraining myeloid-cell-derived S100A8/A9.

GIP regulates inflammation and body weight by restraining myeloid-cell-derived S100A8/A9.

Journal: Nature metabolism

Published: June 07, 2018

Hepatic Amiodarone Lipotoxicity Is Ameliorated by Genetic and Pharmacological Inhibition of Endoplasmatic Reticulum Stress.

Hepatic Amiodarone Lipotoxicity Is Ameliorated by Genetic and Pharmacological Inhibition of Endoplasmatic Reticulum Stress.

Journal: Toxicological sciences : an official journal of the Society of Toxicology

Published: October 01, 2017

Glucose-Dependent Insulinotropic Polypeptide Receptor Deficiency Leads to Impaired Bone Marrow Hematopoiesis.

Glucose-Dependent Insulinotropic Polypeptide Receptor Deficiency Leads to Impaired Bone Marrow Hematopoiesis.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: August 18, 2016

Predictors for advanced fibrosis in morbidly obese non-alcoholic fatty liver patients.

Predictors for advanced fibrosis in morbidly obese non-alcoholic fatty liver patients.

Journal: World journal of hepatology

Published: August 10, 2016

Sortilin Deficiency Reduces Ductular Reaction, Hepatocyte Apoptosis, and Liver Fibrosis in Cholestatic-Induced Liver Injury.

Sortilin Deficiency Reduces Ductular Reaction, Hepatocyte Apoptosis, and Liver Fibrosis in Cholestatic-Induced Liver Injury.

Journal: The American journal of pathology

Published: April 25, 2016

Long-acting glucose-dependent insulinotropic polypeptide ameliorates obesity-induced adipose tissue inflammation.

Long-acting glucose-dependent insulinotropic polypeptide ameliorates obesity-induced adipose tissue inflammation.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: September 14, 2014

Discriminatory metabolic and inflammatory parameters in serum and omental adipose tissue of obese patients with different insulin sensitivity.

Discriminatory metabolic and inflammatory parameters in serum and omental adipose tissue of obese patients with different insulin sensitivity.

Journal: Journal of clinical & translational endocrinology

Published: June 01, 2014

Showing 1-12 of 16

Last Updated: 09/26/2026