Ivan Gallotta
Naples, Italy
Last Updated: 09/26/2026
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Found 11 publications
Individual C. elegans neurons display differential sensitivity to smn- 1 silencing.
Journal: Life science alliance
Published: January 28, 2026
A new Caenorhabditis elegans model to study copper toxicity in Wilson disease.
Journal: Traffic (Copenhagen, Denmark)
Published: July 27, 2023
TGS1 impacts snRNA 3'-end processing, ameliorates survival motor neuron-dependent neurological phenotypes in vivo and prevents neurodegeneration.
Journal: Nucleic acids research
Published: June 29, 2022
Extracellular proteostasis prevents aggregation during pathogenic attack.
Journal: Nature
Published: October 18, 2018
Automated screening of C. elegans neurodegeneration mutants enabled by microfluidics and image analysis algorithms.
Journal: Integrative biology : quantitative biosciences from nano to macro
Published: August 18, 2018
Silencing of Syntaxin 1A in the Dopaminergic Neurons Decreases the Activity of the Dopamine Transporter and Prevents Amphetamine-Induced Behaviors in C. elegans.
Journal: Frontiers in physiology
Published: January 19, 2018
Methods to Study Changes in Inherent Protein Aggregation with Age in Caenorhabditis elegans.
Journal: Journal of visualized experiments : JoVE
Published: December 30, 2017
Functional Dysregulation of CDC42 Causes Diverse Developmental Phenotypes.
Journal: American journal of human genetics
Published: October 18, 2017
WDR79/TCAB1 plays a conserved role in the control of locomotion and ameliorates phenotypic defects in SMA models.
Journal: Neurobiology of disease
Published: January 18, 2017
Neuron-specific knock-down of SMN1 causes neuron degeneration and death through an apoptotic mechanism.
Journal: Human molecular genetics
Published: December 17, 2015
Showing 1-11 of 11
Last Updated: 09/26/2026