Izabella A. Pawluczyk

Leicester, ENG, United Kingdom

Last Updated: 09/26/2026

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Found 21 publications

B cell-derived exosomal miR-483-5p and its potential role in promoting kidney function loss in IgA nephropathy.

B cell-derived exosomal miR-483-5p and its potential role in promoting kidney function loss in IgA nephropathy.

Journal: Kidney international
Published: July 27, 2023

Clinical application of microRNAs in glomerular diseases.

Clinical application of microRNAs in glomerular diseases.

Journal: Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
Published: April 22, 2022

The Non-Coding RNA Landscape in IgA Nephropathy-Where Are We in 2021?

The Non-Coding RNA Landscape in IgA Nephropathy-Where Are We in 2021?

Journal: Journal of clinical medicine
Published: April 22, 2021

Hepatic cysteine sulphinic acid decarboxylase depletion and defective taurine metabolism in a rat partial nephrectomy model of chronic kidney disease.

Hepatic cysteine sulphinic acid decarboxylase depletion and defective taurine metabolism in a rat partial nephrectomy model of chronic kidney disease.

Journal: BMC nephrology
Published: February 03, 2021

MicroRNA-23b-3p Deletion Induces an IgA Nephropathy-like Disease Associated with Dysregulated Mucosal IgA Synthesis.

MicroRNA-23b-3p Deletion Induces an IgA Nephropathy-like Disease Associated with Dysregulated Mucosal IgA Synthesis.

Journal: Journal of the American Society of Nephrology : JASN
Published: January 31, 2021

A Pilot Study to Predict Risk of IgA Nephropathy Progression Based on miR-204 Expression.

A Pilot Study to Predict Risk of IgA Nephropathy Progression Based on miR-204 Expression.

Journal: Kidney international reports
Published: January 19, 2021

Differential expression of microRNA miR-150-5p in IgA nephropathy as a potential mediator and marker of disease progression.

Differential expression of microRNA miR-150-5p in IgA nephropathy as a potential mediator and marker of disease progression.

Journal: Kidney international
Published: March 05, 2020

Macrophage interactions with collecting duct epithelial cells are capable of driving tubulointerstitial inflammation and fibrosis in immunoglobulin A nephropathy.

Macrophage interactions with collecting duct epithelial cells are capable of driving tubulointerstitial inflammation and fibrosis in immunoglobulin A nephropathy.

Journal: Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
Published: September 25, 2019

Vesicles bearing gifts: the functional importance of micro-RNA transfer in extracellular vesicles in chronic kidney disease.

Vesicles bearing gifts: the functional importance of micro-RNA transfer in extracellular vesicles in chronic kidney disease.

Journal: American journal of physiology. Renal physiology
Published: August 16, 2018

MicroRNAs: a new avenue to understand, investigate and treat immunoglobulin A nephropathy?

MicroRNAs: a new avenue to understand, investigate and treat immunoglobulin A nephropathy?

Journal: Clinical kidney journal
Published: June 12, 2017

β1,4-galactosyltransferase 1 is a novel receptor for IgA in human mesangial cells.

β1,4-galactosyltransferase 1 is a novel receptor for IgA in human mesangial cells.

Journal: Kidney international
Published: September 07, 2016

Sialic acid supplementation ameliorates puromycin aminonucleoside nephrosis in rats.

Sialic acid supplementation ameliorates puromycin aminonucleoside nephrosis in rats.

Journal: Laboratory investigation; a journal of technical methods and pathology
Published: September 29, 2014
Showing 1-12 of 21

Last Updated: 09/26/2026