J B. De Souza

London, ENG, United Kingdom

Last Updated: 09/26/2026

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Found 13 publications

Plasmodium berghei NK65 in Combination with IFN-γ Induces Endothelial Glucocorticoid Resistance via Sustained Activation of p38 and JNK.

Plasmodium berghei NK65 in Combination with IFN-γ Induces Endothelial Glucocorticoid Resistance via Sustained Activation of p38 and JNK.

Journal: Frontiers in immunology

Published: July 04, 2017

Host Resistance to Plasmodium-Induced Acute Immune Pathology Is Regulated by Interleukin-10 Receptor Signaling.

Host Resistance to Plasmodium-Induced Acute Immune Pathology Is Regulated by Interleukin-10 Receptor Signaling.

Journal: Infection and immunity

Published: November 11, 2016

A quantitative brain map of experimental cerebral malaria pathology.

A quantitative brain map of experimental cerebral malaria pathology.

Journal: PLoS pathogens

Published: November 08, 2016

Parasite-Specific CD4+ IFN-γ+ IL-10+ T Cells Distribute within Both Lymphoid and Nonlymphoid Compartments and Are Controlled Systemically by Interleukin-27 and ICOS during Blood-Stage Malaria Infection.

Parasite-Specific CD4+ IFN-γ+ IL-10+ T Cells Distribute within Both Lymphoid and Nonlymphoid Compartments and Are Controlled Systemically by Interleukin-27 and ICOS during Blood-Stage Malaria Infection.

Journal: Infection and immunity

Published: August 26, 2015

IL-18-induced expression of high-affinity IL-2R on murine NK cells is essential for NK-cell IFN-γ production during murine Plasmodium yoelii infection.

IL-18-induced expression of high-affinity IL-2R on murine NK cells is essential for NK-cell IFN-γ production during murine Plasmodium yoelii infection.

Journal: European journal of immunology

Published: August 19, 2015

C-reactive protein is essential for innate resistance to pneumococcal infection.

C-reactive protein is essential for innate resistance to pneumococcal infection.

Journal: Immunology

Published: January 03, 2014

IL-27 receptor signaling regulates memory CD4+ T cell populations and suppresses rapid inflammatory responses during secondary malaria infection.

IL-27 receptor signaling regulates memory CD4+ T cell populations and suppresses rapid inflammatory responses during secondary malaria infection.

Journal: Infection and immunity

Published: October 09, 2013

Protective immunity against malaria after vaccination.

Protective immunity against malaria after vaccination.

Journal: Parasite immunology

Published: September 27, 2013

WSX-1 signalling inhibits CD4⁺ T cell migration to the liver during malaria infection by repressing chemokine-independent pathways.

WSX-1 signalling inhibits CD4⁺ T cell migration to the liver during malaria infection by repressing chemokine-independent pathways.

Journal: PloS one

Published: June 18, 2013

IL-27 receptor signaling regulates CD4+ T cell chemotactic responses during infection.

IL-27 receptor signaling regulates CD4+ T cell chemotactic responses during infection.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: March 29, 2013

IL-27 receptor signalling restricts the formation of pathogenic, terminally differentiated Th1 cells during malaria infection by repressing IL-12 dependent signals.

IL-27 receptor signalling restricts the formation of pathogenic, terminally differentiated Th1 cells during malaria infection by repressing IL-12 dependent signals.

Journal: PLoS pathogens

Published: October 19, 2012

IFN-γ-producing CD4+ T cells promote experimental cerebral malaria by modulating CD8+ T cell accumulation within the brain.

IFN-γ-producing CD4+ T cells promote experimental cerebral malaria by modulating CD8+ T cell accumulation within the brain.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: June 23, 2012

Showing 1-12 of 13

Last Updated: 09/26/2026