Jae R. Lim
Seoul, Republic of Korea
Last Updated: 09/26/2026
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Found 19 publications
Folate receptor 1 activation suppresses high glucose-induced amyloidogenesis in neurons via STAT3/Nrf2 pathway-dependent mitigation of mitochondrial oxidative stress.
Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie
Published: October 20, 2025
Recovery of FAM134A-mediated ER-phagy through BRD4 inhibition alleviates ethanol-induced neurodegeneration.
Journal: International journal of biological sciences
Published: April 29, 2025
Restoration of Miro1's N-terminal GTPase function alleviates prenatal stress-induced mitochondrial fission via Drp1 modulation.
Journal: Cell communication and signaling : CCS
Published: January 06, 2025
Sodium butyrate ameliorates high glucose-suppressed neuronal mitophagy by restoring PRKN expression via inhibiting the RELA-HDAC8 complex.
Journal: Autophagy
Published: February 27, 2024
TRIM16-mediated lysophagy suppresses high-glucose-accumulated neuronal Aβ.
Journal: Autophagy
Published: June 26, 2023
Ethanol-induced ceramide production causes neuronal apoptosis by increasing MCL-1S-mediated ER-mitochondria contacts.
Journal: Neurobiology of disease
Published: December 13, 2022
Melatonin-mediated FKBP4 downregulation protects against stress-induced neuronal mitochondria dysfunctions by blocking nuclear translocation of GR.
Journal: Cell death & disease
Published: October 17, 2022
Cyanidin 3-O-arabinoside suppresses DHT-induced dermal papilla cell senescence by modulating p38-dependent ER-mitochondria contacts.
Journal: Journal of biomedical science
Published: November 09, 2021
High glucose-mediated VPS26a down-regulation dysregulates neuronal amyloid precursor protein processing and tau phosphorylation.
Journal: British journal of pharmacology
Published: May 28, 2021
Melatonin activates ABCA1 via the BiP/NRF1 pathway to suppress high-cholesterol-induced apoptosis of mesenchymal stem cells.
Journal: Stem cell research & therapy
Published: August 31, 2020
BNIP3L/NIX-mediated mitophagy protects against glucocorticoid-induced synapse defects.
Journal: Nature communications
Published: June 10, 2020
Urolithin A suppresses high glucose-induced neuronal amyloidogenesis by modulating TGM2-dependent ER-mitochondria contacts and calcium homeostasis.
Journal: Cell death and differentiation
Published: March 09, 2020
Last Updated: 09/26/2026