Jan P. Delling

Albert Einstein Allee 11
Ulm, BW 89081, Germany

Last Updated: 09/26/2026

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Found 10 publications

Neuropeptide CRH prevents premature differentiation of OPCs following CNS injury and in early postnatal development.

Neuropeptide CRH prevents premature differentiation of OPCs following CNS injury and in early postnatal development.

Journal: Cell reports

Published: January 25, 2025

Combined expansion and STED microscopy reveals altered fingerprints of postsynaptic nanostructure across brain regions in ASD-related SHANK3-deficiency.

Combined expansion and STED microscopy reveals altered fingerprints of postsynaptic nanostructure across brain regions in ASD-related SHANK3-deficiency.

Journal: Molecular psychiatry

Published: October 05, 2023

Cacna1c deficiency in forebrain glutamatergic neurons alters behavior and hippocampal plasticity in female mice.

Cacna1c deficiency in forebrain glutamatergic neurons alters behavior and hippocampal plasticity in female mice.

Journal: Translational psychiatry

Published: July 29, 2023

Elevation of SHANK3 Levels by Antisense Oligonucleotides Directed Against the 3'-UTR of the Human SHANK3 mRNA.

Elevation of SHANK3 Levels by Antisense Oligonucleotides Directed Against the 3'-UTR of the Human SHANK3 mRNA.

Journal: Nucleic acid therapeutics

Published: November 10, 2022

Development of sex- and genotype-specific behavioral phenotypes in a Shank3 mouse model for neurodevelopmental disorders.

Development of sex- and genotype-specific behavioral phenotypes in a Shank3 mouse model for neurodevelopmental disorders.

Journal: Frontiers in behavioral neuroscience

Published: September 22, 2022

SHANK3 deficiency leads to myelin defects in the central and peripheral nervous system.

SHANK3 deficiency leads to myelin defects in the central and peripheral nervous system.

Journal: Cellular and molecular life sciences : CMLS

Published: January 31, 2022

Comparison of SHANK3 deficiency in animal models: phenotypes, treatment strategies, and translational implications.

Comparison of SHANK3 deficiency in animal models: phenotypes, treatment strategies, and translational implications.

Journal: Journal of neurodevelopmental disorders

Published: July 15, 2021

Phelan McDermid Syndrome: Multiple Sclerosis as a Rare but Treatable Cause for Regression-A Case Report.

Phelan McDermid Syndrome: Multiple Sclerosis as a Rare but Treatable Cause for Regression-A Case Report.

Journal: International journal of molecular sciences

Published: January 17, 2021

FUS Mislocalization and Vulnerability to DNA Damage in ALS Patients Derived hiPSCs and Aging Motoneurons.

FUS Mislocalization and Vulnerability to DNA Damage in ALS Patients Derived hiPSCs and Aging Motoneurons.

Journal: Frontiers in cellular neuroscience

Published: August 18, 2016

The Shank3 Interaction Partner ProSAPiP1 Regulates Postsynaptic SPAR Levels and the Maturation of Dendritic Spines in Hippocampal Neurons.

The Shank3 Interaction Partner ProSAPiP1 Regulates Postsynaptic SPAR Levels and the Maturation of Dendritic Spines in Hippocampal Neurons.

Journal: Frontiers in synaptic neuroscience

Published: February 04, 2016

Showing 1-10 of 10

Last Updated: 09/26/2026