Jane E. Murphy
Parkville, VIC 3010, Australia
Last Updated: 09/26/2026
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Found 9 publications
The Pyroptotic Cell Death Effector Gasdermin D Is Activated by Gout-Associated Uric Acid Crystals but Is Dispensable for Cell Death and IL-1β Release.
Journal: Journal of immunology (Baltimore, Md. : 1950)
Published: February 27, 2019
G-CSF and Neutrophils Are Nonredundant Mediators of Murine Experimental Autoimmune Uveoretinitis.
Journal: The American journal of pathology
Published: February 05, 2015
Endothelin-converting enzyme 1 and β-arrestins exert spatiotemporal control of substance P-induced inflammatory signals.
Journal: The Journal of biological chemistry
Published: June 06, 2014
Proteolytic activation of the human epithelial sodium channel by trypsin IV and trypsin I involves distinct cleavage sites.
Journal: The Journal of biological chemistry
Published: May 21, 2014
Agonist-biased trafficking of somatostatin receptor 2A in enteric neurons.
Journal: The Journal of biological chemistry
Published: August 06, 2013
The bile acid receptor TGR5 does not interact with β-arrestins or traffic to endosomes but transmits sustained signals from plasma membrane rafts.
Journal: The Journal of biological chemistry
Published: July 03, 2013
Flow cytometric analysis of STAT phosphorylation.
Journal: Methods in molecular biology (Clifton, N.J.)
Published: January 09, 2013
Proteolytic activation of the epithelial sodium channel (ENaC) by the cysteine protease cathepsin-S.
Proteolytic activation of the epithelial sodium channel (ENaC) by the cysteine protease cathepsin-S.
Journal: Pflugers Archiv : European journal of physiology
Published: April 30, 2012
Neurotensin-induced proinflammatory signaling in human colonocytes is regulated by β-arrestins and endothelin-converting enzyme-1-dependent endocytosis and resensitization of neurotensin receptor 1.
Journal: The Journal of biological chemistry
Published: March 15, 2012
Showing 1-9 of 9
Last Updated: 09/26/2026