Last Updated: 09/26/2026
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Found 13 publications
Cardiac fibroblasts counterbalance cardiomyocytes in LMNA cardiomyopathy pathogenesis.
Journal: bioRxiv : the preprint server for biology
Published: July 22, 2025
Perinuclear damage from nuclear envelope deterioration elicits stress responses that contribute to LMNA cardiomyopathy.
Journal: bioRxiv : the preprint server for biology
Published: February 24, 2023
Med25 Limits Master Regulators That Govern Adipogenesis.
Journal: International journal of molecular sciences
Published: February 20, 2023
Elevated dual specificity protein phosphatase 4 in cardiomyopathy caused by lamin A/C gene mutation is primarily ERK1/2-dependent and its depletion improves cardiac function and survival.
Journal: Human molecular genetics
Published: January 22, 2018
Nuclear envelope regulation of signaling cascades.
Journal: Advances in experimental medicine and biology
Published: February 25, 2014
Inhibition of extracellular signal-regulated kinase 1/2 signaling has beneficial effects on skeletal muscle in a mouse model of Emery-Dreifuss muscular dystrophy caused by lamin A/C gene mutation.
Journal: Skeletal muscle
Published: January 03, 2013
Dual specificity phosphatase 4 mediates cardiomyopathy caused by lamin A/C (LMNA) gene mutation.
Journal: The Journal of biological chemistry
Published: October 11, 2012
Reactivation of autophagy ameliorates LMNA cardiomyopathy.
Journal: Autophagy
Published: October 10, 2012
Temsirolimus activates autophagy and ameliorates cardiomyopathy caused by lamin A/C gene mutation.
Journal: Science translational medicine
Published: July 28, 2012
Last Updated: 09/26/2026