Jeanne M. James

Pediatric Cardiology

3333 Burnet Ave
Cincinnati,, OH 45229

Last Updated: 09/26/2026

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Found 8 publications

MYH6 Variants Are Associated with Atrial Dysfunction in Neonates with Hypoplastic Left Heart Syndrome.

MYH6 Variants Are Associated with Atrial Dysfunction in Neonates with Hypoplastic Left Heart Syndrome.

Journal: Genes

Published: September 10, 2024

Significance of α-Myosin Heavy Chain (MYH6) Variants in Hypoplastic Left Heart Syndrome and Related Cardiovascular Diseases.

Significance of α-Myosin Heavy Chain (MYH6) Variants in Hypoplastic Left Heart Syndrome and Related Cardiovascular Diseases.

Journal: Journal of cardiovascular development and disease

Published: April 06, 2022

A mutant fibrinogen that is unable to form fibrin can improve renal phenotype in mice with sickle cell anemia.

A mutant fibrinogen that is unable to form fibrin can improve renal phenotype in mice with sickle cell anemia.

Journal: EJHaem

Published: March 26, 2021

Reply to Angé et al.

Reply to Angé et al.

Journal: American journal of physiology. Heart and circulatory physiology

Published: April 05, 2019

Elimination of the fibrinogen integrin αMβ2-binding motif improves renal pathology in mice with sickle cell anemia.

Elimination of the fibrinogen integrin αMβ2-binding motif improves renal pathology in mice with sickle cell anemia.

Journal: Blood advances

Published: January 26, 2019

Age-dependent cardiac function during experimental sepsis: effect of pharmacological activation of AMP-activated protein kinase by AICAR.

Age-dependent cardiac function during experimental sepsis: effect of pharmacological activation of AMP-activated protein kinase by AICAR.

Journal: American journal of physiology. Heart and circulatory physiology

Published: July 07, 2018

Loss of Axin2 results in impaired heart valve maturation and subsequent myxomatous valve disease.

Loss of Axin2 results in impaired heart valve maturation and subsequent myxomatous valve disease.

Journal: Cardiovascular research

Published: May 06, 2016

Conditional deletion of cardiomyocyte peroxisome proliferator-activated receptor γ enhances myocardial ischemia-reperfusion injury in mice.

Conditional deletion of cardiomyocyte peroxisome proliferator-activated receptor γ enhances myocardial ischemia-reperfusion injury in mice.

Journal: Shock (Augusta, Ga.)

Published: October 04, 2013

Showing 1-8 of 8

Last Updated: 09/26/2026