Jennifer Stanic
Milan, Italy
Last Updated: 09/26/2026
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Found 16 publications
Missense variants in RPH3A cause defects in excitatory synaptic function and are associated with a clinically variable neurodevelopmental disorder.
Journal: Genetics in medicine : official journal of the American College of Medical Genetics
Published: October 19, 2022
Rabphilin-3A Drives Structural Modifications of Dendritic Spines Induced by Long-Term Potentiation.
Journal: Cells
Published: April 19, 2022
GluA3 autoantibodies induce alterations in dendritic spine and behavior in mice.
Journal: Brain, behavior, and immunity
Published: March 29, 2021
Linking NMDA Receptor Synaptic Retention to Synaptic Plasticity and Cognition.
Journal: iScience
Published: April 25, 2019
NMDA receptor GluN2D subunit participates to levodopa-induced dyskinesia pathophysiology.
Journal: Neurobiology of disease
Published: July 18, 2018
Publisher Correction: Anti-AMPA GluA3 antibodies in Frontotemporal dementia: a new molecular target.
Publisher Correction: Anti-AMPA GluA3 antibodies in Frontotemporal dementia: a new molecular target.
Journal: Scientific reports
Published: January 07, 2018
Rabphilin 3A: A novel target for the treatment of levodopa-induced dyskinesias.
Journal: Neurobiology of disease
Published: May 01, 2017
Anti-AMPA GluA3 antibodies in Frontotemporal dementia: a new molecular target.
Journal: Scientific reports
Published: January 25, 2017
LRRK2 phosphorylation level correlates with abnormal motor behaviour in an experimental model of levodopa-induced dyskinesias.
Journal: Molecular brain
Published: February 22, 2016
Last Updated: 09/26/2026