Jenny M. Gunnersen

Melbourne, VIC, Australia

Last Updated: 09/26/2026

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Found 25 publications

Correction: Sez6L2 inhibits complement by facilitating factor I cleavage of C3b and accelerating the decay of C3 convertases.

Correction: Sez6L2 inhibits complement by facilitating factor I cleavage of C3b and accelerating the decay of C3 convertases.

Journal: Frontiers in immunology

Published: May 04, 2026

SEZ6L2 Loss Disrupts Motor Coordination, Cognitive Function, and Synaptic Connectivity.

SEZ6L2 Loss Disrupts Motor Coordination, Cognitive Function, and Synaptic Connectivity.

Journal: bioRxiv : the preprint server for biology

Published: January 08, 2026

Transforming Chronic Pain Management: Integrating Neuromodulation with Advanced Technologies to Tackle Cognitive Dysfunction - A Narrative Review.

Transforming Chronic Pain Management: Integrating Neuromodulation with Advanced Technologies to Tackle Cognitive Dysfunction - A Narrative Review.

Journal: Journal of pain research

Published: January 21, 2025

The BACE1-generated C-terminal fragment of the neural cell adhesion molecule 2 (NCAM2) promotes BACE1 targeting to Rab11-positive endosomes.

The BACE1-generated C-terminal fragment of the neural cell adhesion molecule 2 (NCAM2) promotes BACE1 targeting to Rab11-positive endosomes.

Journal: Cellular and molecular life sciences : CMLS

Published: June 15, 2022

The β-Secretase Substrate Seizure 6-Like Protein (SEZ6L) Controls Motor Functions in Mice.

The β-Secretase Substrate Seizure 6-Like Protein (SEZ6L) Controls Motor Functions in Mice.

Journal: Molecular neurobiology

Published: August 11, 2021

BACE inhibitor treatment of mice induces hyperactivity in a Seizure-related gene 6 family dependent manner without altering learning and memory.

BACE inhibitor treatment of mice induces hyperactivity in a Seizure-related gene 6 family dependent manner without altering learning and memory.

Journal: Scientific reports

Published: April 06, 2021

The Sez6 Family Inhibits Complement by Facilitating Factor I Cleavage of C3b and Accelerating the Decay of C3 Convertases.

The Sez6 Family Inhibits Complement by Facilitating Factor I Cleavage of C3b and Accelerating the Decay of C3 Convertases.

Journal: Frontiers in immunology

Published: September 17, 2020

Evidence that a defined population of neurons in lateral amygdala is directly involved in auditory fear learning and memory.

Evidence that a defined population of neurons in lateral amygdala is directly involved in auditory fear learning and memory.

Journal: Neurobiology of learning and memory

Published: November 29, 2019

Seizure protein 6 controls glycosylation and trafficking of kainate receptor subunits GluK2 and GluK3.

Seizure protein 6 controls glycosylation and trafficking of kainate receptor subunits GluK2 and GluK3.

Journal: The EMBO journal

Published: September 14, 2019

Lack of Sez6 Family Proteins Impairs Motor Functions, Short-Term Memory, and Cognitive Flexibility and Alters Dendritic Spine Properties.

Lack of Sez6 Family Proteins Impairs Motor Functions, Short-Term Memory, and Cognitive Flexibility and Alters Dendritic Spine Properties.

Journal: Cerebral cortex (New York, N.Y. : 1991)

Published: June 14, 2019

Sez6 levels are elevated in cerebrospinal fluid of patients with inflammatory pain-associated conditions.

Sez6 levels are elevated in cerebrospinal fluid of patients with inflammatory pain-associated conditions.

Journal: Pain reports

Published: September 22, 2018

Neurons Specifically Activated by Fear Learning in Lateral Amygdala Display Increased Synaptic Strength.

Neurons Specifically Activated by Fear Learning in Lateral Amygdala Display Increased Synaptic Strength.

Journal: eNeuro

Published: March 25, 2018

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Last Updated: 09/26/2026