Jens Schuster

Uppsala, C, Sweden

Last Updated: 09/26/2026

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Found 34 publications

Plasma proteomic profile reveals persistent immune activation in post-acute sequelae of SARS-CoV-2 infection.

Plasma proteomic profile reveals persistent immune activation in post-acute sequelae of SARS-CoV-2 infection.

Journal: Frontiers in immunology
Published: December 24, 2025

Abnormalities in the functional activity of neural networks in a human iPSC model of Dravet syndrome.

Abnormalities in the functional activity of neural networks in a human iPSC model of Dravet syndrome.

Journal: Neuroscience research
Published: June 26, 2025

Comprehensive transcriptome assessment in PBMCs of post-COVID patients at a median follow-up of 28 months after a mild COVID infection reveals upregulation of JAK/STAT signaling and a prolonged immune response.

Comprehensive transcriptome assessment in PBMCs of post-COVID patients at a median follow-up of 28 months after a mild COVID infection reveals upregulation of JAK/STAT signaling and a prolonged immune response.

Journal: Frontiers in immunology
Published: March 11, 2025

Epigenetic insights into GABAergic development in Dravet Syndrome iPSC and therapeutic implications.

Epigenetic insights into GABAergic development in Dravet Syndrome iPSC and therapeutic implications.

Journal: eLife
Published: August 27, 2024

KRIT1 heterozygous mutations are sufficient to induce a pathological phenotype in patient-derived iPSC models of cerebral cavernous malformation.

KRIT1 heterozygous mutations are sufficient to induce a pathological phenotype in patient-derived iPSC models of cerebral cavernous malformation.

Journal: Cell reports
Published: December 09, 2023

Generation of a ZEB2 deficient human iPSC line (KICRi002A-4).

Generation of a ZEB2 deficient human iPSC line (KICRi002A-4).

Journal: Stem cell research
Published: December 06, 2023

ZEB2 haploinsufficient Mowat-Wilson syndrome induced pluripotent stem cells show disrupted GABAergic transcriptional regulation and function.

ZEB2 haploinsufficient Mowat-Wilson syndrome induced pluripotent stem cells show disrupted GABAergic transcriptional regulation and function.

Journal: Frontiers in molecular neuroscience
Published: July 07, 2022

Monoallelic and bi-allelic variants in NCDN cause neurodevelopmental delay, intellectual disability, and epilepsy.

Monoallelic and bi-allelic variants in NCDN cause neurodevelopmental delay, intellectual disability, and epilepsy.

Journal: American journal of human genetics
Published: March 04, 2022

Generation of a human iPSC line (UUIGPi015-A) from a patient with Dravet syndrome and a 2.9 Mb deletion spanning SCN1A on chromosome 2.

Generation of a human iPSC line (UUIGPi015-A) from a patient with Dravet syndrome and a 2.9 Mb deletion spanning SCN1A on chromosome 2.

Journal: Stem cell research
Published: January 14, 2022

Syndromic RNA polymerase II insufficiency: Generation of a human induced pluripotent stem cell line (UUIGPi002A-5) with a heterozygous disruption of POLR2A.

Syndromic RNA polymerase II insufficiency: Generation of a human induced pluripotent stem cell line (UUIGPi002A-5) with a heterozygous disruption of POLR2A.

Journal: Stem cell research
Published: September 07, 2021

Monoallelic and bi-allelic variants in NCDN cause neurodevelopmental delay, intellectual disability, and epilepsy.

Monoallelic and bi-allelic variants in NCDN cause neurodevelopmental delay, intellectual disability, and epilepsy.

Journal: American journal of human genetics
Published: November 26, 2020

Generation of human induced pluripotent stem cell (iPSC) lines (UUMCBi001-A, UUMCBi002-A) from two healthy donors.

Generation of human induced pluripotent stem cell (iPSC) lines (UUMCBi001-A, UUMCBi002-A) from two healthy donors.

Journal: Stem cell research
Published: November 21, 2020
Showing 1-12 of 34

Last Updated: 09/26/2026