Jessica N. Andrade
Belo Horizonte, MG, Brazil
Last Updated: 09/26/2026
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Found 9 publications
Increased oxidative stress and CaMKII activity contribute to electro-mechanical defects in cardiomyocytes from a murine model of Huntington's disease.
Journal: The FEBS journal
Published: August 29, 2018
Corrigendum to "Neuromuscular synapse degeneration without muscle function loss in the diaphragm of a murine model for Huntington's Disease" [Neurochem. Int. 116 (2018) 30-42].
Journal: Neurochemistry international
Published: May 28, 2018
Fast and slow-twitching muscles are differentially affected by reduced cholinergic transmission in mice deficient for VAChT: A mouse model for congenital myasthenia.
Journal: Neurochemistry international
Published: February 13, 2018
Neuromuscular synapse degeneration without muscle function loss in the diaphragm of a murine model for Huntington's Disease.
Journal: Neurochemistry international
Published: October 20, 2017
Muscle atrophy is associated with cervical spinal motoneuron loss in BACHD mouse model for Huntington's disease.
Journal: The European journal of neuroscience
Published: April 18, 2016
The mGluR5 positive allosteric modulator, CDPPB, ameliorates pathology and phenotypic signs of a mouse model of Huntington's disease.
Journal: Neurobiology of disease
Published: May 09, 2014
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Last Updated: 09/26/2026