Ji-hong Moon

Jeonju, Republic of Korea

Last Updated: 09/26/2026

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Found 31 publications

HIF-1α stabilization inhibits Japanese encephalitis virus propagation and neurotoxicity via autophagy pathways.

HIF-1α stabilization inhibits Japanese encephalitis virus propagation and neurotoxicity via autophagy pathways.

Journal: Biochemical and biophysical research communications

Published: October 16, 2024

Calcium-dependent serine-threonine phosphatase and autophagy inactivation mediated by Baicalein attenuates prion protein-mediated neuronal cell damage.

Calcium-dependent serine-threonine phosphatase and autophagy inactivation mediated by Baicalein attenuates prion protein-mediated neuronal cell damage.

Journal: BMC complementary medicine and therapies

Published: February 16, 2024

Melatonin-mediated calcineurin inactivation attenuates amyloid beta-induced apoptosis.

Melatonin-mediated calcineurin inactivation attenuates amyloid beta-induced apoptosis.

Journal: IBRO neuroscience reports

Published: August 29, 2023

Antiviral activity of prion protein against Japanese encephalitis virus infection in vitro and in vivo.

Antiviral activity of prion protein against Japanese encephalitis virus infection in vitro and in vivo.

Journal: Virus research

Published: June 09, 2023

Melatonin inhibits Japanese encephalitis virus replication and neurotoxicity via calcineurin-autophagy pathways.

Melatonin inhibits Japanese encephalitis virus replication and neurotoxicity via calcineurin-autophagy pathways.

Journal: BMC neuroscience

Published: November 02, 2022

Calcineurin, Calcium-Dependent Serine-Threonine Phosphatase Activation by Prion Peptide 106-126 Enhances Nuclear Factor-κB-Linked Proinflammatory Response through Autophagy Pathway.

Calcineurin, Calcium-Dependent Serine-Threonine Phosphatase Activation by Prion Peptide 106-126 Enhances Nuclear Factor-κB-Linked Proinflammatory Response through Autophagy Pathway.

Journal: ACS chemical neuroscience

Published: August 23, 2021

Calcineurin Activation by Prion Protein Induces Neurotoxicity via Mitochondrial Reactive Oxygen Species.

Calcineurin Activation by Prion Protein Induces Neurotoxicity via Mitochondrial Reactive Oxygen Species.

Journal: Oxidative medicine and cellular longevity

Published: January 28, 2021

The antidiabetic drug troglitazone protects against PrP (106‑126)‑induced neurotoxicity via the PPARγ‑autophagy pathway in neuronal cells.

The antidiabetic drug troglitazone protects against PrP (106‑126)‑induced neurotoxicity via the PPARγ‑autophagy pathway in neuronal cells.

Journal: Molecular medicine reports

Published: September 04, 2020

Prion peptide-mediated calcium level alteration governs neuronal cell damage through AMPK-autophagy flux.

Prion peptide-mediated calcium level alteration governs neuronal cell damage through AMPK-autophagy flux.

Journal: Cell communication and signaling : CCS

Published: February 13, 2020

Human prion protein-mediated calcineurin activation induces neuron cell death via AMPK and autophagy pathway.

Human prion protein-mediated calcineurin activation induces neuron cell death via AMPK and autophagy pathway.

Journal: The international journal of biochemistry & cell biology

Published: September 20, 2019

Inhibition of Autophagy by Captopril Attenuates Prion Peptide-Mediated Neuronal Apoptosis via AMPK Activation.

Inhibition of Autophagy by Captopril Attenuates Prion Peptide-Mediated Neuronal Apoptosis via AMPK Activation.

Journal: Molecular neurobiology

Published: June 14, 2018

Telmisartan generates ROS-dependent upregulation of death receptor 5 to sensitize TRAIL in lung cancer via inhibition of autophagy flux.

Telmisartan generates ROS-dependent upregulation of death receptor 5 to sensitize TRAIL in lung cancer via inhibition of autophagy flux.

Journal: The international journal of biochemistry & cell biology

Published: April 26, 2018

Showing 1-12 of 31

Last Updated: 09/26/2026