Ji-wei Hao

51 Fucheng Road
Haidian, 100048, China

Last Updated: 09/26/2026

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Found 11 publications

Interleukin-33/IL-1RL1 axis modulates dendritic cell immune responses during the early phase of sepsis via RETREG1-mediated reticulophagy.

Interleukin-33/IL-1RL1 axis modulates dendritic cell immune responses during the early phase of sepsis via RETREG1-mediated reticulophagy.

Journal: Journal of advanced research

Published: March 06, 2026

Hyperuricemia was associated with metabolic response against brain injury instead of metabolism syndrome in Tibet: a cross-sectional analysis.

Hyperuricemia was associated with metabolic response against brain injury instead of metabolism syndrome in Tibet: a cross-sectional analysis.

Journal: Frontiers in endocrinology

Published: December 27, 2025

Citrus pectin protects mice from burn injury by modulating intestinal microbiota, GLP-1 secretion and immune response.

Citrus pectin protects mice from burn injury by modulating intestinal microbiota, GLP-1 secretion and immune response.

Journal: International immunopharmacology

Published: January 07, 2024

Single-cell transcriptome profiling of sepsis identifies HLA-DRlowS100Ahigh monocytes with immunosuppressive function.

Single-cell transcriptome profiling of sepsis identifies HLA-DRlowS100Ahigh monocytes with immunosuppressive function.

Journal: Military Medical Research

Published: December 13, 2022

Central GLP-1 Resistance Induced by Severe Traumatic Brain Injury Was Associated with Persistent Hyperglycemia in Humans.

Central GLP-1 Resistance Induced by Severe Traumatic Brain Injury Was Associated with Persistent Hyperglycemia in Humans.

Journal: Neuroendocrinology

Published: November 04, 2022

Prognostic Significance of Plasma Insulin Level for Deep Venous Thrombosis in Patients with Severe Traumatic Brain Injury in Critical Care.

Prognostic Significance of Plasma Insulin Level for Deep Venous Thrombosis in Patients with Severe Traumatic Brain Injury in Critical Care.

Journal: Neurocritical care

Published: March 25, 2022

Exendin-4 Exacerbates Burn-Induced Mortality in Mice by Switching to Th2 Response.

Exendin-4 Exacerbates Burn-Induced Mortality in Mice by Switching to Th2 Response.

Journal: The Journal of surgical research

Published: February 22, 2022

Plasma glucagon-like peptide 1 was associated with hospital-acquired infections and long-term mortality in burn patients.

Plasma glucagon-like peptide 1 was associated with hospital-acquired infections and long-term mortality in burn patients.

Journal: Surgery

Published: September 15, 2019

Exendin-4 Exacerbates Burn-Induced Morbidity in Mice by Activation of the Sympathetic Nervous System.

Exendin-4 Exacerbates Burn-Induced Morbidity in Mice by Activation of the Sympathetic Nervous System.

Journal: Mediators of inflammation

Published: July 10, 2018

Proinflammatory switch from Gαs to Gαi signaling by Glucagon-like peptide-1 receptor in murine splenic monocyte following burn injury.

Proinflammatory switch from Gαs to Gαi signaling by Glucagon-like peptide-1 receptor in murine splenic monocyte following burn injury.

Journal: Inflammation research : official journal of the European Histamine Research Society ... [et al.]

Published: July 18, 2017

Early antagonism of cerebral high mobility group box-1 protein is benefit for sepsis induced brain injury.

Early antagonism of cerebral high mobility group box-1 protein is benefit for sepsis induced brain injury.

Journal: Oncotarget

Published: July 29, 2016

Showing 1-11 of 11

Last Updated: 09/26/2026