Jianye Peng

Jiefang Road 30
Hengyang, 421000, China

Last Updated: 09/26/2026

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Found 17 publications

Sulfiredoxin 1 ameliorates doxorubicin-induced cardiotoxicity by suppressing oxidative stress and inflammation via the Sirt1/NLRP3 pathway.

Sulfiredoxin 1 ameliorates doxorubicin-induced cardiotoxicity by suppressing oxidative stress and inflammation via the Sirt1/NLRP3 pathway.

Journal: International immunopharmacology

Published: May 05, 2024

Interleukin-5 alleviates cardiac remodelling via the STAT3 pathway in angiotensin II-infused mice.

Interleukin-5 alleviates cardiac remodelling via the STAT3 pathway in angiotensin II-infused mice.

Journal: Journal of cellular and molecular medicine

Published: August 14, 2023

CTRP5 Attenuates Doxorubicin-Induced Cardiotoxicity Via Inhibiting TLR4/NLRP3 Signaling.

CTRP5 Attenuates Doxorubicin-Induced Cardiotoxicity Via Inhibiting TLR4/NLRP3 Signaling.

Journal: Cardiovascular drugs and therapy

Published: May 01, 2023

Upregulation of CIRP by its agonist prevents the development of heart failure in myocardial infarction rats.

Upregulation of CIRP by its agonist prevents the development of heart failure in myocardial infarction rats.

Journal: BMC cardiovascular disorders

Published: March 21, 2023

Crocin alleviates neurotoxicity induced by bupivacaine in SH-SY5Y cells with inhibition of PI3K/AKT signaling.

Crocin alleviates neurotoxicity induced by bupivacaine in SH-SY5Y cells with inhibition of PI3K/AKT signaling.

Journal: Genes & genomics

Published: February 13, 2023

Loss of MD1 Promotes Inflammatory and Apoptotic Atrial Remodelling in Diabetic Cardiomyopathy by Activating the TLR4/NF-κB Signalling Pathway.

Loss of MD1 Promotes Inflammatory and Apoptotic Atrial Remodelling in Diabetic Cardiomyopathy by Activating the TLR4/NF-κB Signalling Pathway.

Journal: Pharmacology

Published: September 19, 2022

Resveratrol protects against ox-LDL-induced endothelial dysfunction in atherosclerosis via depending on circ_0091822/miR-106b-5p-mediated upregulation of TLR4.

Resveratrol protects against ox-LDL-induced endothelial dysfunction in atherosclerosis via depending on circ_0091822/miR-106b-5p-mediated upregulation of TLR4.

Journal: Immunopharmacology and immunotoxicology

Published: June 23, 2022

Mitochondrial derived peptide MOTS-c prevents the development of heart failure under pressure overload conditions in mice.

Mitochondrial derived peptide MOTS-c prevents the development of heart failure under pressure overload conditions in mice.

Journal: Journal of cellular and molecular medicine

Published: February 10, 2022

Correction to: AAV9-Mediated Cardiac CNTF Overexpression Exacerbated Adverse Cardiac Remodeling in Streptozotocin-Induced Type 1 Diabetic Models.

Correction to: AAV9-Mediated Cardiac CNTF Overexpression Exacerbated Adverse Cardiac Remodeling in Streptozotocin-Induced Type 1 Diabetic Models.

Journal: Cardiovascular toxicology

Published: December 08, 2021

The role of Cold-Inducible RNA-binding protein in respiratory diseases.

The role of Cold-Inducible RNA-binding protein in respiratory diseases.

Journal: Journal of cellular and molecular medicine

Published: October 29, 2021

AAV9-Mediated Cardiac CNTF Overexpression Exacerbated Adverse Cardiac Remodeling in Streptozotocin-Induced Type 1 Diabetic Models.

AAV9-Mediated Cardiac CNTF Overexpression Exacerbated Adverse Cardiac Remodeling in Streptozotocin-Induced Type 1 Diabetic Models.

Journal: Cardiovascular toxicology

Published: August 02, 2021

The Mitochondrial-Derived Peptide MOTS-c Attenuates Oxidative Stress Injury and the Inflammatory Response of H9c2 Cells Through the Nrf2/ARE and NF-κB Pathways.

The Mitochondrial-Derived Peptide MOTS-c Attenuates Oxidative Stress Injury and the Inflammatory Response of H9c2 Cells Through the Nrf2/ARE and NF-κB Pathways.

Journal: Cardiovascular engineering and technology

Published: July 14, 2021

Showing 1-12 of 17

Last Updated: 09/26/2026