Jie-sen Zhou

Key Laboratory Of Respiratory Disease Of Zhejiang Province
Hangzhou, 310009, China

Last Updated: 09/26/2026

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Found 17 publications

Neutrophil Heterogeneity in Airway Inflammatory Diseases.

Neutrophil Heterogeneity in Airway Inflammatory Diseases.

Journal: Inflammation

Published: May 20, 2025

Cholesterol Accumulation Enhances Cigarette Smoke-Induced Airway Epithelial Inflammation.

Cholesterol Accumulation Enhances Cigarette Smoke-Induced Airway Epithelial Inflammation.

Journal: International journal of chronic obstructive pulmonary disease

Published: October 12, 2024

The persistent inflammation in COPD: is autoimmunity the core mechanism?

The persistent inflammation in COPD: is autoimmunity the core mechanism?

Journal: European respiratory review : an official journal of the European Respiratory Society

Published: July 06, 2023

Correction: Caveolin-1 inhibits expression of antioxidant enzymes through direct interaction with nuclear erythroid 2 p45-related factor-2 (Nrf2).

Correction: Caveolin-1 inhibits expression of antioxidant enzymes through direct interaction with nuclear erythroid 2 p45-related factor-2 (Nrf2).

Journal: The Journal of biological chemistry

Published: July 26, 2020

Correction: Caveolin-1 inhibits expression of antioxidant enzymes through direct interaction with nuclear erythroid 2 p45-related factor-2 (Nrf2).

Correction: Caveolin-1 inhibits expression of antioxidant enzymes through direct interaction with nuclear erythroid 2 p45-related factor-2 (Nrf2).

Journal: The Journal of biological chemistry

Published: July 12, 2020

Cigarette smoke-initiated autoimmunity facilitates sensitisation to elastin-induced COPD-like pathologies in mice.

Cigarette smoke-initiated autoimmunity facilitates sensitisation to elastin-induced COPD-like pathologies in mice.

Journal: The European respiratory journal

Published: August 15, 2019

Endoplasmic reticulum chaperone GRP78 mediates cigarette smoke-induced necroptosis and injury in bronchial epithelium.

Endoplasmic reticulum chaperone GRP78 mediates cigarette smoke-induced necroptosis and injury in bronchial epithelium.

Journal: International journal of chronic obstructive pulmonary disease

Published: February 16, 2018

MTOR Suppresses Cigarette Smoke-Induced Epithelial Cell Death and Airway Inflammation in Chronic Obstructive Pulmonary Disease.

MTOR Suppresses Cigarette Smoke-Induced Epithelial Cell Death and Airway Inflammation in Chronic Obstructive Pulmonary Disease.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: December 06, 2017

Autophagy inhibitors suppress environmental particulate matter-induced airway inflammation.

Autophagy inhibitors suppress environmental particulate matter-induced airway inflammation.

Journal: Toxicology letters

Published: June 12, 2017

Early growth response factor 1 is essential for cigarette smoke-induced MUC5AC expression in human bronchial epithelial cells.

Early growth response factor 1 is essential for cigarette smoke-induced MUC5AC expression in human bronchial epithelial cells.

Journal: Biochemical and biophysical research communications

Published: May 28, 2017

Efficacy and safety of bronchoscopic lung volume reduction therapy in patients with severe emphysema: a meta-analysis of randomized controlled trials.

Efficacy and safety of bronchoscopic lung volume reduction therapy in patients with severe emphysema: a meta-analysis of randomized controlled trials.

Journal: Oncotarget

Published: February 27, 2017

Neutrophilic Inflammation in the Immune Responses of Chronic Obstructive Pulmonary Disease: Lessons from Animal Models.

Neutrophilic Inflammation in the Immune Responses of Chronic Obstructive Pulmonary Disease: Lessons from Animal Models.

Journal: Journal of immunology research

Published: January 31, 2017

Showing 1-12 of 17

Last Updated: 09/26/2026