Last Updated: 09/26/2026
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Found 61 publications
Severe obesity in human HFpEF alters contractile protein function and organization.
Journal: Science (New York, N.Y.)
Published: April 23, 2026
Mutation-induced free-energy remodeling of recovery stroke and ATP hydrolysis in human cardiac β-myosin.
Journal: Biophysical journal
Published: February 09, 2026
Unraveling Mutation-Induced Protein Communication Pathways in the Actomyosin Complex: Insights from Comprehensive Metadynamics Simulations.
Journal: The journal of physical chemistry. B
Published: August 22, 2025
Resolving the structure and dynamics of the disordered C terminus of human cardiac troponin T and effects of cardiomyopathic mutations.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: July 08, 2025
A rare splice-site variant in TNNT2: the need for ancestral diversity in genomic reference data sets.
Journal: European heart journal
Published: October 18, 2024
Arg92Leu-cTnT Alters the cTnC-cTnI Interface Disrupting PKA-Mediated Relaxation.
Journal: Circulation research
Published: September 27, 2024
The transmission of mutation effects in a multiprotein machine: A comprehensive metadynamics study of the cardiac thin filament.
Journal: Protein science : a publication of the Protein Society
Published: August 29, 2024
Empagliflozin enhances metabolic efficiency and improves left ventricular hypertrophy in a hypertrophic cardiomyopathy mouse model.
Journal: European heart journal
Published: June 25, 2024
Myosin-Catalyzed ATP Hydrolysis in the Presence of Disease-Causing Mutations: Mavacamten as a Way to Repair Mechanism.
Journal: The journal of physical chemistry. B
Published: May 06, 2024
Human cardiac β-myosin powerstroke energetics: Thin filament, Pi displacement, and mutation effects.
Human cardiac β-myosin powerstroke energetics: Thin filament, Pi displacement, and mutation effects.
Journal: Biophysical journal
Published: April 11, 2024
The HCM - Linked Mutation Arg92Leu in TNNT2 Allosterically Alters the cTnC - cTnI Interface and Disrupts the PKA-mediated Regulation of Myofilament Relaxation.
Journal: bioRxiv : the preprint server for biology
Published: July 28, 2023
Divergent Molecular Phenotypes in Point Mutations at the Same Residue in Beta-Myosin Heavy Chain Lead to Distinct Cardiomyopathies.
Journal: bioRxiv : the preprint server for biology
Published: July 18, 2023
Last Updated: 09/26/2026