Jin A. Shin
Seoul, Republic of Korea
Last Updated: 09/26/2026
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Found 16 publications
Noggin-mediated effects on metabolite profiles of microglia and oligodendrocytes after ischemic insult.
Journal: Journal of pharmaceutical and biomedical analysis
Published: July 01, 2022
Enhanced repair processes and iron uptake by ischemic preconditioning in the brain during the recovery phase after ischemic stroke.
Journal: Brain research
Published: July 03, 2020
Iron released from reactive microglia by noggin improves myelin repair in the ischemic brain.
Journal: Neuropharmacology
Published: October 06, 2017
Repression of adenosine triphosphate-binding cassette transporter ABCG2 by estrogen increases intracellular glutathione in brain endothelial cells following ischemic reperfusion injury.
Journal: Neurobiology of aging
Published: August 31, 2017
Resveratrol attenuates peripheral and brain inflammation and reduces ischemic brain injury in aged female mice.
Journal: Neurobiology of aging
Published: September 30, 2015
Activation of classical estrogen receptor subtypes reduces tight junction disruption of brain endothelial cells under ischemia/reperfusion injury.
Journal: Free radical biology & medicine
Published: July 07, 2015
Visceral adipose tissue inflammation is associated with age-related brain changes and ischemic brain damage in aged mice.
Journal: Brain, behavior, and immunity
Published: April 14, 2015
Matrix metalloproteinase-8 plays a pivotal role in neuroinflammation by modulating TNF-α activation.
Matrix metalloproteinase-8 plays a pivotal role in neuroinflammation by modulating TNF-α activation.
Journal: Journal of immunology (Baltimore, Md. : 1950)
Published: July 23, 2014
Estrogen receptor-mediated resveratrol actions on blood-brain barrier of ovariectomized mice.
Journal: Neurobiology of aging
Published: April 10, 2014
Noggin improves ischemic brain tissue repair and promotes alternative activation of microglia in mice.
Journal: Brain, behavior, and immunity
Published: December 04, 2013
Extracellular signal-regulated kinase1/2-dependent changes in tight junctions after ischemic preconditioning contributes to tolerance induction after ischemic stroke.
Journal: Brain structure & function
Published: April 28, 2013
Last Updated: 09/26/2026